Naser Jaleel, Ph.D.
Affiliations: | 2010 | Physiology | Temple University, Philadelphia, PA, United States |
Area:
Physiology Biology, General BiophysicsGoogle:
"Naser Jaleel"Mean distance: (not calculated yet)
Parents
Sign in to add mentor| Steven R. Houser | grad student | 2010 | Temple University | |
| (Re-expression of T-type calcium channels minimally affects cardiac contractility and activates pro-survival signaling pathways in the myocardium.) | ||||
BETA: Related publications
See more...
Publications
|
You can help our author matching system! If you notice any publications incorrectly attributed to this author, please sign in and mark matches as correct or incorrect. |
| Raake PW, Zhang X, Vinge LE, et al. (2012) Cardiac G-protein-coupled receptor kinase 2 ablation induces a novel Ca2+ handling phenotype resistant to adverse alterations and remodeling after myocardial infarction. Circulation. 125: 2108-18 |
| Zhang H, Chen X, Gao E, et al. (2010) Increasing cardiac contractility after myocardial infarction exacerbates cardiac injury and pump dysfunction Circulation Research. 107: 800-809 |
| Kubo H, Berretta RM, Jaleel N, et al. (2009) c-Kit+ bone marrow stem cells differentiate into functional cardiac myocytes. Clinical and Translational Science. 2: 26-32 |
| MacDonnell SM, Weisser-Thomas J, Kubo H, et al. (2009) CaMKII negatively regulates calcineurin-NFAT signaling in cardiac myocytes. Circulation Research. 105: 316-25 |
| Jaleel N, Nakayama H, Chen X, et al. (2008) Ca2+ influx through T- and L-type Ca2+ channels have different effects on myocyte contractility and induce unique cardiac phenotypes. Circulation Research. 103: 1109-19 |
| Kubo H, Jaleel N, Kumarapeli A, et al. (2008) Increased cardiac myocyte progenitors in failing human hearts. Circulation. 118: 649-57 |
| Nakayama H, Chen X, Baines CP, et al. (2007) Ca2+- and mitochondrial-dependent cardiomyocyte necrosis as a primary mediator of heart failure. The Journal of Clinical Investigation. 117: 2431-44 |
| Chen X, Wilson RM, Kubo H, et al. (2007) Adolescent feline heart contains a population of small, proliferative ventricular myocytes with immature physiological properties. Circulation Research. 100: 536-44 |
| Jaleel N, Kubo H, Macdonnell S, et al. (2007) Constitutive Activation of CaMKII Reduces Cardiac Function and Viability in Feline Ventricular Myocytes Journal of Cardiac Failure. 13: S94-S95 |
| Zhang H, Chen X, Gao E, et al. (2007) Excess Ca2+ Influx through the L-Type Ca2+ Channel Exacerbates Ischemia-Induced Cardiac Damage Journal of Cardiac Failure. 13: S87-S88 |