Larry A. Barr, Ph.D.
Affiliations: | 2014 | Physiology | Temple University, Philadelphia, PA, United States |
Area:
Physiology Biology, Molecular Biology, PharmacologyGoogle:
"Larry Barr"Mean distance: (not calculated yet)
Parents
Sign in to add mentorSteven R. Houser | grad student | 2014 | Temple University | |
(The role of calcium in the regulation of pathological hypertrophy.) |
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Publications
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Barr LA, Lambert JP, Shimizu Y, et al. (2017) Exercise training provides cardioprotection by activating and coupling endothelial nitric oxide synthase via a β3-adrenergic receptor-AMP-activated protein kinase signaling pathway. Medical Gas Research. 7: 1-8 |
Shimizu Y, Nicholson CK, Lambert JP, et al. (2016) Sodium Sulfide Attenuates Ischemic-Induced Heart Failure by Enhancing Proteasomal Function in an Nrf2-Dependent Manner. Circulation. Heart Failure. 9 |
Barr LA, Shimizu Y, Lambert JP, et al. (2015) Hydrogen sulfide attenuates high fat diet-induced cardiac dysfunction via the suppression of endoplasmic reticulum stress. Nitric Oxide : Biology and Chemistry / Official Journal of the Nitric Oxide Society. 46: 145-56 |
Shimizu Y, Nicholson CK, Lambert JP, et al. (2015) Hydrogen sulfide therapy attenuates the development of heart failure by increasing the activity of the proteasome in an Nrf2-dependent manner Nitric Oxide. 47: S25 |
Tilley DG, Zhu W, Myers VD, et al. (2014) β-adrenergic receptor-mediated cardiac contractility is inhibited via vasopressin type 1A-receptor-dependent signaling. Circulation. 130: 1800-11 |
Barr LA, Makarewich CA, Berretta RM, et al. (2014) Imatinib activates pathological hypertrophy by altering myocyte calcium regulation. Clinical and Translational Science. 7: 360-7 |
Gao H, Wang F, Wang W, et al. (2012) Ca(2+) influx through L-type Ca(2+) channels and transient receptor potential channels activates pathological hypertrophy signaling. Journal of Molecular and Cellular Cardiology. 53: 657-67 |