Grant A. Bledsoe, Ph.D.
Affiliations: | 2004 | Medical University of South Carolina, Charleston, SC, United States |
Area:
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Parents
Sign in to add mentor| Julie Chao | grad student | 2004 | MUSC | |
| (Tissue kallikrein in cardiac remodeling and renal damage in genetic and salt-induced hypertension rat models.) | ||||
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Publications
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| Guo Y, Li P, Gao L, et al. (2017) Kallistatin reduces vascular senescence and aging by regulating microRNA-34a-SIRT1 pathway. Aging Cell |
| Chao J, Bledsoe G, Chao L. (2016) Protective Role of Kallistatin in Vascular and Organ Injury. Hypertension |
| Li P, Guo Y, Bledsoe G, et al. (2016) Kallistatin Induces Breast Cancer Cell Apoptosis and Autophagy by Modulating Wnt Signaling and microRNA Synthesis. Experimental Cell Research |
| Guo Y, Li P, Bledsoe G, et al. (2015) Kallistatin inhibits TGF-β-induced endothelial-mesenchymal transition by differential regulation of microRNA-21 and eNOS expression. Experimental Cell Research. 337: 103-10 |
| Li P, Guo Y, Bledsoe G, et al. (2015) Kallistatin treatment attenuates lethality and organ injury in mouse models of established sepsis. Critical Care (London, England). 19: 200 |
| Chao J, Bledsoe G, Chao L. (2014) Kallikrein-kinin in stem cell therapy. World Journal of Stem Cells. 6: 448-57 |
| Gao L, Li P, Zhang J, et al. (2014) Novel role of kallistatin in vascular repair by promoting mobility, viability, and function of endothelial progenitor cells. Journal of the American Heart Association. 3: e001194 |
| Chao J, Bledsoe G, Chao L. (2014) Tissue kallikrein-kinin therapy in hypertension and organ damage. Progress in Drug Research. Fortschritte Der Arzneimittelforschung. Progrã¨S Des Recherches Pharmaceutiques. 69: 37-57 |
| Li P, Bledsoe G, Yang ZR, et al. (2014) Human kallistatin administration reduces organ injury and improves survival in a mouse model of polymicrobial sepsis. Immunology. 142: 216-26 |
| Gao L, Bledsoe G, Yin H, et al. (2013) Tissue kallikrein-modified mesenchymal stem cells provide enhanced protection against ischemic cardiac injury after myocardial infarction. Circulation Journal : Official Journal of the Japanese Circulation Society. 77: 2134-44 |