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Jack H. Kaplan

Affiliations: 
University of Illinois at Chicago, Health Sciences Center, Chicago, IL 60612, United States 
Area:
Cell Biology, Molecular Biology, Biochemistry
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Children

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Melissa Laughery grad student 2006 University of Illinois at Chicago, Health Sciences Center
Graham C. R. Ellis-Davies post-doc University of Illinois at Chicago, Health Sciences Center (Neurotree)

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Publications

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Fukai T, Ushio-Fukai M, Kaplan JH. (2018) Copper Transporters and Copper Chaperones: Roles in Cardiovascular Physiology and Disease. American Journal of Physiology. Cell Physiology
Habeck M, Tokhtaeva E, Nadav Y, et al. (2016) Selective assembly of Na,K-ATPase α2β2 heterodimers in the heart: distinct functional properties and isoform-selective inhibitors. The Journal of Biological Chemistry
Clifford RJ, Maryon EB, Kaplan JH. (2016) Dynamic internalization and recycling of a metal ion transporter: Cu homeostasis and hCTR1, the human Cu uptake system. Journal of Cell Science
Kaplan JH, Maryon EB. (2016) How Mammalian Cells Acquire Copper: An Essential but Potentially Toxic Metal. Biophysical Journal. 110: 7-13
Shattock MJ, Ottolia M, Bers DM, et al. (2015) Na+/Ca2+ exchange and Na+/K+-ATPase in the heart. The Journal of Physiology. 593: 1361-82
Clifford RJ, Kaplan JH. (2013) Human breast tumor cells are more resistant to cardiac glycoside toxicity than non-tumorigenic breast cells. Plos One. 8: e84306
Maryon EB, Molloy SA, Ivy K, et al. (2013) Rate and regulation of copper transport by human copper transporter 1 (hCTR1). The Journal of Biological Chemistry. 288: 18035-46
Ivy KD, Kaplan JH. (2013) A re-evaluation of the role of hCTR1, the human high-affinity copper transporter, in platinum-drug entry into human cells. Molecular Pharmacology. 83: 1237-46
Maryon EB, Molloy SA, Kaplan JH. (2013) Cellular glutathione plays a key role in copper uptake mediated by human copper transporter 1. American Journal of Physiology. Cell Physiology. 304: C768-79
Arystarkhova E, Liu YB, Salazar C, et al. (2013) Hyperplasia of pancreatic beta cells and improved glucose tolerance in mice deficient in the FXYD2 subunit of Na,K-ATPase. The Journal of Biological Chemistry. 288: 7077-85
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