Michael R. Blackburn
Affiliations: | The University of Texas Graduate School of Biomedical Sciences at Houston |
Area:
Pathology, BiochemistryGoogle:
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Publications
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Wang W, Chen NY, Ren D, et al. (2021) Enhancing Extracellular Adenosine Levels Restores Barrier Function in Acute Lung Injury Through Expression of Focal Adhesion Proteins. Frontiers in Molecular Biosciences. 8: 636678 |
Chambers ED, White A, Vang A, et al. (2020) Blockade of equilibrative nucleoside transporter 1/2 protects against Pseudomonas aeruginosa-induced acute lung injury and NLRP3 inflammasome activation. Faseb Journal : Official Publication of the Federation of American Societies For Experimental Biology. 34: 1516-1531 |
Weng T, Huang J, Wagner EJ, et al. (2019) Downregulation of CFIm25 amplifies dermal fibrosis through alternative polyadenylation. The Journal of Experimental Medicine |
Ko J, Mills T, Huang J, et al. (2019) Transforming growth factor beta 1 alters the 3'UTR of mRNA to promote lung fibrosis. The Journal of Biological Chemistry |
Wu M, Skaug B, Bi X, et al. (2019) Interferon regulatory factor 7 (IRF7) represents a link between inflammation and fibrosis in the pathogenesis of systemic sclerosis. Annals of the Rheumatic Diseases |
Collum SD, Molina JG, Hanmandlu A, et al. (2019) Adenosine and hyaluronan modulate lung fibrosis and pulmonary hypertension in combined pulmonary fibrosis and emphysema (CPFE). Disease Models & Mechanisms |
Huang C, Xiao X, Yang Y, et al. (2019) Correction: MicroRNA-101 attenuates pulmonary fibrosis by inhibiting fibroblast proliferation and activation. The Journal of Biological Chemistry. 294: 6694 |
Weng T, Ko J, Masamha CP, et al. (2019) Cleavage factor 25 deregulation contributes to pulmonary fibrosis through alternative polyadenylation. The Journal of Clinical Investigation. 130 |
Aherne CM, Collins CB, Rapp CR, et al. (2018) Coordination of ENT2-dependent adenosine transport and signaling dampens mucosal inflammation. Jci Insight. 3 |
Liu H, Adebiyi M, Liu RR, et al. (2018) Elevated ecto-5'-nucleotidase: a missing pathogenic factor and new therapeutic target for sickle cell disease. Blood Advances. 2: 1957-1968 |