Moon-shong Tang
Affiliations: | Environmental Health Science | New York University, New York, NY, United States |
Area:
Environmental Sciences, Oncology, BiochemistryGoogle:
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Publications
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Tang MS, Lee HW, Weng MW, et al. (2021) DNA damage, DNA repair and carcinogenicity: Tobacco smoke versus electronic cigarette aerosol. Mutation Research. Reviews in Mutation Research. 789: 108409 |
Wang HT, Lee HW, Weng MW, et al. (2021) The role of TAp63γ and P53 point mutations in regulating DNA repair, mutational susceptibility and invasion of bladder cancer cells. Elife. 10 |
Pham K, Huynh D, Le L, et al. (2020) E-cigarette promotes breast carcinoma progression and lung metastasis: Macrophage-tumor cells crosstalk and the role of CCL5 and VCAM-1. Cancer Letters |
Tang M, Lee H, Chen L, et al. (2019) Mp63-02 E-Cigarette Smoke Is Potentially Bladder Carcinogenic - It Induces Dna Damage And Urothelial Hyperplasia In Mice The Journal of Urology. 201 |
He F, Zhang F, Liu Y, et al. (2019) Mp51-13 Hyper-Phosphorylation Of Pten Is A Principal Mechanism Of Pten Inactivation In Human Bladder Cancer The Journal of Urology. 201 |
Weng MW, Lee HW, Park SH, et al. (2018) Aldehydes are the predominant forces inducing DNA damage and inhibiting DNA repair in tobacco smoke carcinogenesis. Proceedings of the National Academy of Sciences of the United States of America |
Tang MS. (2018) Reply to Queimado et al.: E-cigarettes induce DNA damage and inhibit DNA repair in mice and human cells. Proceedings of the National Academy of Sciences of the United States of America |
Lee HW, Park SH, Weng MW, et al. (2018) E-cigarette smoke damages DNA and reduces repair activity in mouse lung, heart, and bladder as well as in human lung and bladder cells. Proceedings of the National Academy of Sciences of the United States of America |
Wang HT, Lin JH, Yang CH, et al. (2017) Acrolein induces mtDNA damages, mitochondrial fission and mitophagy in human lung cells. Oncotarget. 8: 70406-70421 |
Weng MW, Lee HW, Choi B, et al. (2017) AFB1 hepatocarcinogenesis is via lipid peroxidation that inhibits DNA repair, sensitizes mutation susceptibility and induces aldehyde-DNA adducts at p53 mutational hotspot codon 249. Oncotarget |