Michael S. Wolin
Affiliations: | Physiology | New York Medical College, New York, NY, United States |
Area:
Physiology Biology, BiochemistryWebsite:
https://www.nymc.edu/faculty/directory/by-name/wolin-michael/Google:
"Michael S. Wolin"Bio:
https://www.linkedin.com/in/mike-wolin-ab026280
http://www.jbc.org/content/255/2/331.short
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Parents
Sign in to add mentor| John A. Gerlt | grad student | 1981 | Yale | |
| (Adenylate cyclase from Brevibacterium liquefaciens A kinetic characterization of the catalytic reaction and regulation of the enzyme by uncomplexed Mg++ and pyruvate.) | ||||
| Louis J. Ignarro | post-doc | 1983 | Tulane University Medical School | |
Children
Sign in to add trainee| Warren H. Lee | grad student | 2001 | New York Medical College |
| Richard A. Oeckler | grad student | 2004 | New York Medical College |
| Christopher J. Mingone | grad student | 2005 | New York Medical College |
| Mansoor Ahmad | grad student | 2007 | New York Medical College |
| Qun Gao | grad student | 2008 | New York Medical College |
| Raed Alhawaj | grad student | 2014 | New York Medical College |
BETA: Related publications
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Publications
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| Yu H, Alruwaili N, Kelly MR, et al. (2022) Endothelin-1 Depletion of Cartilage Oligomeric Matrix Protein Modulates Pulmonary Artery Superoxide and Iron Metabolism-associated Mitochondrial Heme Biosynthesis. American Journal of Physiology. Lung Cellular and Molecular Physiology |
| Katseff A, Alhawaj R, Wolin MS. (2021) Redox and Inflammatory Signaling, the Unfolded Protein Response, and the Pathogenesis of Pulmonary Hypertension. Advances in Experimental Medicine and Biology. 1304: 333-373 |
| Hashimoto R, Lanier GM, Dhagia V, et al. (2020) Pluripotent HSCs Augment a-Adrenergic Receptor-Mediated Contraction of Pulmonary Artery and Contribute to the Pathogenesis of Pulmonary Hypertension. American Journal of Physiology. Lung Cellular and Molecular Physiology |
| Alruwaili N, Kandhi S, Kelly MR, et al. (2020) Increased extracellular superoxide and MMP9 attenuated COMP stabilization of BMPR2 potentially participate in pulmonary hypertension development The Faseb Journal. 34: 1-1 |
| Kandhi S, Alruwaili N, Wolin MS, et al. (2019) Reciprocal actions of constrictor prostanoids and superoxide in chronic hypoxia-induced pulmonary hypertension: roles of EETs. Pulmonary Circulation. 9: 2045894019895947 |
| Yu H, Alruwaili N, Hu B, et al. (2019) Potential Role of Cartilage Oligomeric Matrix Protein in the Modulation of Pulmonary Arterial Smooth Muscle Superoxide by Hypoxia. American Journal of Physiology. Lung Cellular and Molecular Physiology |
| Wolin MS, Alruwaili N, Kandhi S. (2019) Studies on Hypoxic Pulmonary Vasoconstriction Detect a Novel Role for the Mitochondrial Complex I Subunit Ndufs2 in Controlling Peroxide Generation for Oxygen-Sensing. Circulation Research. 124: 1683-1685 |
| Alruwaili N, Kandhi S, Sun D, et al. (2018) Metabolism and Redox in Pulmonary Vascular Physiology and Pathophysiology. Antioxidants & Redox Signaling |
| Zhang B, Alruwaili N, Kandhi S, et al. (2018) Inhibition of ferrochelatase impairs vascular eNOS/NO and sGC/cGMP signaling. Plos One. 13: e0200307 |
| Patel D, Lakhkar A, Wolin MS. (2017) Redox Mechanisms Influencing cGMP Signaling in Pulmonary Vascular Physiology and Pathophysiology. Advances in Experimental Medicine and Biology. 967: 227-240 |