Sabine Hellwig, Ph.D.
Affiliations: | 2007 | University of Utah, Salt Lake City, UT |
Area:
Molecular BiologyGoogle:
"Sabine Hellwig"Mean distance: (not calculated yet)
Parents
Sign in to add mentorBrenda L. Bass | grad student | 2007 | University of Utah | |
(The fate of edited RNA in Caenorhabditis elegans.) |
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Publications
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Weir MC, Shu ST, Patel RK, et al. (2018) Selective inhibition of the myeloid Src-family kinase Fgr potently suppresses AML cell growth in vitro and in vivo. Acs Chemical Biology |
Weir MC, Hellwig S, Tan L, et al. (2017) Dual inhibition of Fes and Flt3 tyrosine kinases potently inhibits Flt3-ITD+ AML cell growth. Plos One. 12: e0181178 |
Weir MC, Shu S, Hellwig S, et al. (2017) A Unique N-Phenylbenzamide Inhibitor Targeting Non-Receptor Tyrosine Kinases Potently Suppresses AML Cell Growth In Vitro and In Vivo Blood. 130: 3813-3813 |
Weir M, Hellwig S, Gray NS, et al. (2015) Abstract A25: Dual inhibition of Flt3 and Fes tyrosine kinases potently blocks proliferation of AML cells expressing an active Flt3 mutant. Clinical Cancer Research. 21 |
Weir MC, Hellwig S, Patel R, et al. (2015) Abstract B180: Multi-targeted tyrosine kinase inhibitors potently suppress FLT3-ITD+ AML cell growth Molecular Cancer Therapeutics. 14 |
Weir M, Hellwig S, Gray NS, et al. (2013) Abstract 2086: Dual inhibitors of Flt3 and c-Fest tyrosine kinases potently inhibit proliferation of AML cells expressing an active Flt3 mutant. Cancer Research. 73: 2086-2086 |
Hellwig S, Miduturu CV, Kanda S, et al. (2012) Small-molecule inhibitors of the c-Fes protein-tyrosine kinase. Chemistry & Biology. 19: 529-40 |
Hellwig S, Smithgall TE. (2011) Structure and regulation of the c-Fes protein-tyrosine kinase. Frontiers in Bioscience (Landmark Edition). 16: 3146-55 |
Hellwig S, Gray NS, Smithgall TE. (2011) Abstract A234: Identification and characterization of inhibitor-resistant mutants of the protein-tyrosine kinase c-Fes. Molecular Cancer Therapeutics. 10 |
Aruscavage PJ, Hellwig S, Bass BL. (2010) Small DNA pieces in C. elegans are intermediates of DNA fragmentation during apoptosis. Plos One. 5: e11217 |