Sanford I. Bernstein
Affiliations: | University of California, San Diego and San Diego State University |
Area:
Cell Biology, Biochemistry, Molecular BiologyGoogle:
"Sanford Bernstein"Mean distance: (not calculated yet)
Children
Sign in to add trainee| Kevin Edwards | grad student | 1987-1990 | (Cell Biology Tree) |
| James T. Caldwell | post-doc | 2013-2018 |
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Publications
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| Kronert WA, Hsu KH, Madan A, et al. (2022) Myosin Transducer Inter-Strand Communication Is Critical for Normal ATPase Activity and Myofibril Structure. Biology. 11 |
| Trujillo AS, Hsu KH, Viswanathan MC, et al. (2022) The R369 Myosin Residue within Loop 4 Is Critical for Actin Binding and Muscle Function in . International Journal of Molecular Sciences. 23 |
| Trujillo AS, Hsu KH, Puthawala J, et al. (2021) Myosin dilated cardiomyopathy mutation S532P disrupts actomyosin interactions, leading to altered muscle kinetics, reduced locomotion, and cardiac dilation in . Molecular Biology of the Cell. mbcE21020088 |
| Bell KM, Huang A, Kronert WA, et al. (2021) Prolonged Myosin Binding Increases Muscle Stiffness in Drosophila Models of Freeman-Sheldon Syndrome. Biophysical Journal |
| Bloemink MJ, Hsu KH, Geeves MA, et al. (2020) Alternative N-terminal regions of myosin heavy chain II regulate communication of the purine binding loop with the essential light chain. The Journal of Biological Chemistry |
| Guo Y, Kronert WA, Hsu KH, et al. (2020) Drosophila myosin mutants model the disparate severity of type 1 and type 2B distal arthrogryposis and indicate an enhanced actin affinity mechanism. Skeletal Muscle. 10: 24 |
| Suggs JA, Manalo K, Melkani GC, et al. (2020) Manipulating Levels of Stress‐Response Proteins in a Drosophila Model of Myosin‐Based Inclusion Body Myopathy 3 Worsens Muscle Dysfunction The Faseb Journal. 34: 1-1 |
| Caldwell JT, Mermelstein DJ, Walker RC, et al. (2019) X-ray crystallographic and molecular dynamic analyses of Drosophila melanogaster embryonic muscle myosin define domains responsible for isoform-specific properties. Journal of Molecular Biology |
| Bell KM, Kronert WA, Huang A, et al. (2019) The R249Q hypertrophic cardiomyopathy myosin mutation decreases contractility in Drosophila by impeding force production. The Journal of Physiology |
| Rao DS, Kronert WA, Guo Y, et al. (2018) Reductions in ATPase activity, actin sliding velocity and myofibril stability yield muscle dysfunction in Drosophila models of myosin-based Freeman Sheldon syndrome. Molecular Biology of the Cell. mbcE18080526 |