Claire Bastie
Affiliations: | 2004-2007 | University of Iowa, Iowa City, IA |
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| Tarabra E, An Lee TW, Zammit VA, et al. (2017) Differential activation of Fyn kinase distinguishes saturated and unsaturated fats in mouse macrophages. Oncotarget. 8: 86634-86645 |
| Yamada E, Okada S, Bastie CC, et al. (2016) Fyn phosphorylates AMPK to inhibit AMPK activity and AMP-dependent activation of autophagy. Oncotarget |
| Yamada E, Okada S, Shibusawa R, et al. (2016) Identifying a new pathway to regulate AMPK activity under metabolic stress Diabetes Research and Clinical Practice. 120: S180 |
| Apontes P, Liu Z, Su K, et al. (2014) Mangiferin stimulates carbohydrate oxidation and protects against metabolic disorders induced by high-fat diets. Diabetes. 63: 3626-36 |
| Yang Y, Tarabra E, Yang GS, et al. (2013) Alteration of de novo glucose production contributes to fasting hypoglycaemia in Fyn deficient mice. Plos One. 8: e81866 |
| Kuliawat R, Klein L, Gong Z, et al. (2013) Potent humanin analog increases glucose-stimulated insulin secretion through enhanced metabolism in the β cell. Faseb Journal : Official Publication of the Federation of American Societies For Experimental Biology. 27: 4890-8 |
| Martinez-Lopez N, Athonvarangkul D, Sahu S, et al. (2013) Autophagy in Myf5+ progenitors regulates energy and glucose homeostasis through control of brown fat and skeletal muscle development. Embo Reports. 14: 795-803 |
| Lee TW, Kwon H, Zong H, et al. (2013) Fyn deficiency promotes a preferential increase in subcutaneous adipose tissue mass and decreased visceral adipose tissue inflammation. Diabetes. 62: 1537-46 |
| Yamada E, Bastie CC, Koga H, et al. (2012) Mouse skeletal muscle fiber-type-specific macroautophagy and muscle wasting are regulated by a Fyn/STAT3/Vps34 signaling pathway. Cell Reports. 1: 557-69 |
| Bastie CC, Gaffney-Stomberg E, Lee TW, et al. (2012) Dietary cholecalciferol and calcium levels in a Western-style defined rodent diet alter energy metabolism and inflammatory responses in mice. The Journal of Nutrition. 142: 859-65 |