Arnold Schwartz, Ph.D.

Affiliations: 
Institute of Molecular Pharmacology and Biophysics University of Cincinnati, Cincinnati, OH 
Area:
Pharmacology
Google:
"Arnold Schwartz"
Bio:

http://healthnews.uc.edu/news/?/20301/

Mean distance: 18.86 (cluster 28)
 
SNBCP
Cross-listing: Neurotree

BETA: Related publications

Publications

You can help our author matching system! If you notice any publications incorrectly attributed to this author, please sign in and mark matches as correct or incorrect.

Mastrolia V, Flucher SM, Obermair GJ, et al. (2017) Loss of α2δ-1 Calcium Channel Subunit Function Increases the Susceptibility for Diabetes. Diabetes
Margas W, Ferron L, Nieto-Rostro M, et al. (2016) Effect of knockout of α2δ-1 on action potentials in mouse sensory neurons. Philosophical Transactions of the Royal Society of London. Series B, Biological Sciences. 371
Patel R, Bauer CS, Nieto-Rostro M, et al. (2013) α2δ-1 gene deletion affects somatosensory neuron function and delays mechanical hypersensitivity in response to peripheral nerve damage. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 33: 16412-26
Petrashevskaya NN, Ishii M, D'Souza K, et al. (2011) Presynaptic stimulus-release and postsynaptic compensatory changes in mice lacking the N-type calcium channel α(1B)-subunit. Autonomic Neuroscience : Basic & Clinical. 160: 9-15
Nakayama H, Bodi I, Correll RN, et al. (2009) alpha1G-dependent T-type Ca2+ current antagonizes cardiac hypertrophy through a NOS3-dependent mechanism in mice. The Journal of Clinical Investigation. 119: 3787-96
Beetz N, Hein L, Meszaros J, et al. (2009) Transgenic simulation of human heart failure-like L-type Ca2+-channels: implications for fibrosis and heart rate in mice. Cardiovascular Research. 84: 396-406
Fuller-Bicer GA, Varadi G, Koch SE, et al. (2009) Targeted disruption of the voltage-dependent calcium channel alpha2/delta-1-subunit. American Journal of Physiology. Heart and Circulatory Physiology. 297: H117-24
Jagatheesan G, Rajan S, Schulz EM, et al. (2009) An internal domain of beta-tropomyosin increases myofilament Ca(2+) sensitivity. American Journal of Physiology. Heart and Circulatory Physiology. 297: H181-90
Wang S, Ziman B, Bodi I, et al. (2009) Dilated cardiomyopathy with increased SR Ca2+ loading preceded by a hypercontractile state and diastolic failure in the alpha(1C)TG mouse. Plos One. 4: e4133
Jagatheesan G, Schulz EM, Ahmed RP, et al. (2009) An Internal Domain of Beta Tropomyosin Increases Myofilament Calcium Sensitivity Biophysical Journal. 96: 504a
See more...