Giorgio Lagna
Affiliations: | University of California, San Francisco, San Francisco, CA |
Area:
RNA processing, transcriptional regulation, smooth muscle biology, vertebrate developmentGoogle:
"Giorgio Lagna"Mean distance: (not calculated yet)
Collaborators
Sign in to add collaborator| Akiko Hata | collaborator | 1992- | UCSF |
| Joan Massague | collaborator | 1992- | Weill Cornell Medical College (MathTree) |
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Publications
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| Prabhakar A, Kumar R, Wadhwa M, et al. (2023) Reversal of pulmonary veno-occlusive disease phenotypes by inhibition of the integrated stress response. Biorxiv : the Preprint Server For Biology |
| Prabhakar A, Hu S, Tang J, et al. (2023) Essential role of the amino-terminal region of Drosha for the Microprocessor function. Iscience. 26: 107971 |
| Hata A, Lagna G. (2021) How do you mend inactive tumor suppressor mutants? You glue them! Cell Chemical Biology. 28: 585-587 |
| Jiang X, Prabhakar A, Van der Voorn SM, et al. (2021) Control of ribosomal protein synthesis by the Microprocessor complex. Science Signaling. 14 |
| Hata A, Lagna G. (2019) Deregulation of Drosha in the pathogenesis of hereditary hemorrhagic telangiectasia. Current Opinion in Hematology |
| Vattulainen-Collanus S, Southwood M, Yang XD, et al. (2018) Bone morphogenetic protein signaling is required for RAD51-mediated maintenance of genome integrity in vascular endothelial cells. Communications Biology. 1: 149 |
| Jiang X, Wooderchak-Donahue WL, McDonald J, et al. (2018) Inactivating mutations in Drosha mediate vascular abnormalities similar to hereditary hemorrhagic telangiectasia. Science Signaling. 11 |
| Jiang X, Hawkins JS, Lee J, et al. (2017) Let-7 microRNA-dependent control of leukotriene signaling regulates the transition of hematopoietic niche in mice. Nature Communications. 8: 128 |
| Kashima R, Redmond PL, Ghatpande P, et al. (2017) Hyperactive locomotion in a Drosophila model is a functional readout for the synaptic abnormalities underlying fragile X syndrome. Science Signaling. 10 |
| Kashima R, Roy S, Ascano M, et al. (2016) Augmented noncanonical BMP type II receptor signaling mediates the synaptic abnormality of fragile X syndrome. Science Signaling. 9: ra58 |