Deborah F. Wilsker, Ph.D.
Affiliations: | 2005 | Temple University, Philadelphia, PA, United States |
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"Deborah Wilsker"Parents
Sign in to add mentorLi An | grad student | 2005 | Temple University | |
(The DNA binding properties of p270, a member of human SWI /SNF-related complexes and of the ARID family of proteins.) |
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Takebe N, Coyne GHO, Kummar S, et al. (2018) Safety, tolerability, and antitumor activity of once-daily Wee-1 inhibitor AZD1775. Journal of Clinical Oncology. 36: 2587-2587 |
O'Sullivan Coyne GH, Do KT, Kummar S, et al. (2018) Phase I trial of the triplet M6620 (formerly VX970) + veliparib + cisplatin in patients with advanced solid tumors. Journal of Clinical Oncology. 36: 2549-2549 |
O'Sullivan Coyne GH, Kummar S, Meehan RS, et al. (2017) Phase I trial of the triplet veliparib + VX-970 + cisplatin in patients with advanced solid tumors. Journal of Clinical Oncology. 35: TPS2609-TPS2609 |
Meehan RS, Chen AP, Coyne GO, et al. (2017) Abstract 4678: Pilot trial of talazoparib (BMN 673), an oral PARP inhibitor, in patients with advanced solid tumors carrying deleterious BRCA mutations Cancer Research. 77: 4678-4678 |
Wilsker D, Marrero AM, Dull A, et al. (2016) Abstract B24: Ataxia-telangiectasia and Rad3-related (ATR) phosphorylation as a pharmacodynamic biomarker of ATR activation in solid tumor tissue models Molecular Cancer Research. 14 |
Wilsker D, Chung JH, Bunz F. (2012) Chk1 suppresses bypass of mitosis and tetraploidization in p53-deficient cancer cells. Cell Cycle (Georgetown, Tex.). 11: 1564-72 |
Wilsker D, Chung JH, Pradilla I, et al. (2012) Targeted mutations in the ATR pathway define agent-specific requirements for cancer cell growth and survival. Molecular Cancer Therapeutics. 11: 98-107 |
Wilsker DF, Chung JH, Bunz F. (2011) Abstract LB-188: Chk1 suppresses bypass of mitosis and tetraploidization in p53-deficient cells Cancer Research. 71 |
Wilsker D, Bunz F. (2009) Chk1 phosphorylation during mitosis: a new role for a master regulator. Cell Cycle (Georgetown, Tex.). 8: 1161-3 |
Wilsker D, Petermann E, Helleday T, et al. (2008) Essential function of Chk1 can be uncoupled from DNA damage checkpoint and replication control. Proceedings of the National Academy of Sciences of the United States of America. 105: 20752-7 |