Sahar Nissim, Ph.D.

Affiliations: 
Harvard University, Cambridge, MA, United States 
Area:
genetics, morphogenesis, limb, evolution
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"Sahar Nissim"
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Publications

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Aney KJ, Jeong WJ, Vallejo AF, et al. (2024) A Novel Approach for Pancreas Transcriptomics Reveals the Cellular Landscape in Homeostasis and Acute Pancreatitis. Gastroenterology
Tzouanas CN, Sherman MS, Shay JES, et al. (2023) Chronic metabolic stress drives developmental programs and loss of tissue functions in non-transformed liver that mirror tumor states and stratify survival. Biorxiv : the Preprint Server For Biology
Chittenden A, Haraldsdottir S, Chen E, et al. (2022) Germline Testing for Individuals with Pancreatic Adenocarcinoma and Novel Genetic Risk Factors. Hematology/Oncology Clinics of North America. 36: 943-960
Aney KJ, Nissim S. (2021) More than acinar identity? A novel cystic phenotype suggests broader roles for NR5A2 in pancreatic cancer†. The Journal of Pathology
Nissim S, Leshchiner I, Mancias JD, et al. (2019) Mutations in RABL3 alter KRAS prenylation and are associated with hereditary pancreatic cancer. Nature Genetics
Nissim S, Leshchiner I, Mancias JD, et al. (2017) Abstract 4272: Mutations inRABL3alter RAS prenylation and are associated with hereditary pancreatic cancer Epidemiology. 77: 4272-4272
Cox AG, Tsomides A, Kim AJ, et al. (2016) Selenoprotein H is an essential regulator of redox homeostasis that cooperates with p53 in development and tumorigenesis. Proceedings of the National Academy of Sciences of the United States of America
Nissim S, Weeksb O, Talbot JC, et al. (2016) Iterative use of nuclear receptor Nr5a2 regulates multiple stages of liver and pancreas development. Developmental Biology
Cox AG, Hwang KL, Brown KK, et al. (2016) Yap reprograms glutamine metabolism to increase nucleotide biosynthesis and enable liver growth. Nature Cell Biology
Mancias JD, Pontano Vaites L, Nissim S, et al. (2015) Ferritinophagy via NCOA4 is required for erythropoiesis and is regulated by iron dependent HERC2-mediated proteolysis. Elife. 4
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