Brigitte Laforest, Ph.D.
Affiliations: | 2011 | Université de Montréal, Montréal, Canada |
Area:
Hox genes, axis formationGoogle:
"Brigitte Laforest"Parents
Sign in to add mentorMarie Kmita | grad student | 2011 | Université de Montréal | |
(Essential role of GATA5 in the mammalian heart.) |
BETA: Related publications
See more...
Publications
You can help our author matching system! If you notice any publications incorrectly attributed to this author, please sign in and mark matches as correct or incorrect. |
Broman MT, Nadadur RD, Perez-Cervantes C, et al. (2024) A Genomic Link From Heart Failure to Atrial Fibrillation Risk: FOG2 Modulates a TBX5/GATA4-Dependent Atrial Gene Regulatory Network. Circulation |
Barefield DY, Yamakawa S, Tahtah I, et al. (2022) Partial and complete loss of myosin binding protein H-like cause cardiac conduction defects. Journal of Molecular and Cellular Cardiology. 169: 28-40 |
Stefanovic S, Laforest B, Desvignes JP, et al. (2020) -dependent coordination of mouse cardiac progenitor cell patterning and differentiation. Elife. 9 |
Dai W, Nadadur RD, Brennan J, et al. (2020) ZO-1 Regulates Intercalated Disc Composition and Atrioventricular Node Conduction. Circulation Research |
Laforest B, Dai W, Tyan L, et al. (2019) Atrial fibrillation risk loci interact to modulate Ca2+-dependent atrial rhythm homeostasis. The Journal of Clinical Investigation |
Dai W, Laforest B, Tyan L, et al. (2019) A calcium transport mechanism for atrial fibrillation in Tbx5-mutant mice. Elife. 8 |
Dai W, Laforest B, Tyan L, et al. (2019) Author response: A calcium transport mechanism for atrial fibrillation in Tbx5-mutant mice Elife |
Barefield DY, Yamakawa S, Tahtah I, et al. (2019) Abstract 331: Loss of Myosin Binding Protein H-Like Causes Cardiac Conduction Abnormalities Circulation Research. 125 |
Tyan L, Dai W, Nadadur R, et al. (2018) Mechanisms of AP Prolongation and Triggered Activity in a TBX5 Model of Atrial Fibrillation Biophysical Journal. 114 |
Roux M, Laforest B, Eudes N, et al. (2016) Hoxa1 and Hoxb1 are required for pharyngeal arch artery development. Mechanisms of Development |