Didier Picard
Affiliations: | Cell Biology | University of Geneva, Geneva, Switzerland |
Area:
Estrogen ReceptorGoogle:
"Didier Picard"Parents
Sign in to add mentorWalter Schaffner | grad student | University of Zurich (UZH) (FlyTree) | |
Keith R. Yamamoto | post-doc | (Chemistry Tree) |
Children
Sign in to add traineeValentina Gburcik | grad student | 2000-2005 | University of Geneva |
Dina Hany | grad student | 2017-2022 | University of Geneva (Cell Biology Tree) |
Valentina Gburcik | post-doc | 2005-2006 | University of Geneva |
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Publications
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Maiti S, Bhattacharya K, Wider D, et al. (2025) Correction: Hsf1 and the molecular chaperone Hsp90 support a 'rewiring stress response' leading to an adaptive cell size increase in chronic stress. Elife. 14 |
Maiti S, Bhattacharya K, Wider D, et al. (2023) Hsf1 and the molecular chaperone Hsp90 support a 'rewiring stress response' leading to an adaptive cell size increase in chronic stress. Elife. 12 |
Hany D, Vafeiadou V, Picard D. (2023) CRISPR-Cas9 screen reveals a role of purine synthesis for estrogen receptor α activity and tamoxifen resistance of breast cancer cells. Science Advances. 9: eadd3685 |
Hany D, Zoetemelk M, Bhattacharya K, et al. (2023) Network-informed discovery of multidrug combinations for ERα+/HER2-/PI3Kα-mutant breast cancer. Cellular and Molecular Life Sciences : Cmls. 80: 80 |
Bhattacharya K, Maiti S, Zahoran S, et al. (2022) Translational reprogramming in response to accumulating stressors ensures critical threshold levels of Hsp90 for mammalian life. Nature Communications. 13: 6271 |
Mohammadi Ghahhari N, Sznurkowska MK, Hulo N, et al. (2022) Cooperative interaction between ERα and the EMT-inducer ZEB1 reprograms breast cancer cells for bone metastasis. Nature Communications. 13: 2104 |
Vafeiadou V, Hany D, Picard D. (2022) Hyperactivation of MAPK Induces Tamoxifen Resistance in SPRED2-Deficient ERα-Positive Breast Cancer. Cancers. 14 |
Segala G, Bennesch MA, Ghahhari NM, et al. (2019) Vps11 and Vps18 of Vps-C membrane traffic complexes are E3 ubiquitin ligases and fine-tune signalling. Nature Communications. 10: 1833 |
Echeverria PC, Bhattacharya K, Joshi A, et al. (2019) The sensitivity to Hsp90 inhibitors of both normal and oncogenically transformed cells is determined by the equilibrium between cellular quiescence and activity. Plos One. 14: e0208287 |
Berto M, Jean V, Zwart W, et al. (2018) ERα activity depends on interaction and target site corecruitment with phosphorylated CREB1. Life Science Alliance. 1: e201800055 |