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Anne Joutel

Affiliations: 
Université Paris 5 
Area:
Notch3 signaling, Small vessel disease of the brain, Smooth muscle cell, Transcriptome, Myogenic response,
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"Anne Joutel"
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Marie Monet grad student 2008 Université Paris 5
Charles Fouillade grad student 2012 Université Paris 5

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Publications

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Dupré N, Gueniot F, Domenga-Denier V, et al. (2024) Protein aggregates containing wild-type and mutant NOTCH3 are major drivers of arterial pathology in CADASIL. The Journal of Clinical Investigation
Oka F, Lee JH, Yuzawa I, et al. (2022) CADASIL mutations sensitize the brain to ischemia via spreading depolarizations and abnormal extracellular potassium homeostasis. The Journal of Clinical Investigation
Ratelade J, Klug NR, Lombardi D, et al. (2020) Reducing Hypermuscularization of the Transitional Segment between Arterioles and Capillaries Protects Against Spontaneous Intracerebral Hemorrhage. Circulation
Neves KB, Harvey AP, Moreton F, et al. (2019) ER stress and Rho kinase activation underlie the vasculopathy of CADASIL. Jci Insight
Morris HE, Neves KB, Nilsen M, et al. (2019) Abstract P145: Notch3 Mutation is Involved in Pulmonary Vascular Dysfunction and Development of Pulmonary Arterial Hypertension Hypertension. 74
Ghezali L, Capone C, Baron-Menguy C, et al. (2018) Notch3 immunotherapy improves cerebrovascular responses in CADASIL mice. Annals of Neurology
Zellner A, Scharrer E, Arzberger T, et al. (2018) CADASIL brain vessels show a HTRA1 loss-of-function profile. Acta Neuropathologica
Frösen J, Joutel A. (2018) Smooth muscle cells of intracranial vessels: From development to disease. Cardiovascular Research
Ratelade J, Mezouar N, Domenga-Denier V, et al. (2017) Severity of arterial defects in the retina correlates with the burden of intracerebral haemorrhage in COL4A1-related stroke. The Journal of Pathology
Baron-Menguy C, Domenga-Denier V, Ghezali L, et al. (2016) Increased Notch3 Activity Mediates Pathological Changes in Structure of Cerebral Arteries. Hypertension (Dallas, Tex. : 1979)
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