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Elizabeth J. Rossin, Ph.D.

Affiliations: 
2012 Biology: Medical Sciences, Division of Harvard University, Cambridge, MA, United States 
Area:
Genetics
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"Elizabeth Rossin"

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Joel N. Hirschhorn grad student 2012 Harvard
 (The proteomic landscape of human disease: construction and evaluation of networks associated to complex traits.)
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Publications

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Pollack S, Igo RP, Jensen RA, et al. (2018) Multiethnic Genome-wide Association Study of Diabetic Retinopathy using Liability Threshold Modeling of Duration of Diabetes and Glycemic Control. Diabetes
van der Harst P, van Setten J, Verweij N, et al. (2016) 52 Genetic Loci Influencing Myocardial Mass. Journal of the American College of Cardiology. 68: 1435-48
Lundby A, Rossin EJ, Steffensen AB, et al. (2014) Annotation of loci from genome-wide association studies using tissue-specific quantitative interaction proteomics. Nature Methods. 11: 868-74
Arking DE, Pulit SL, Crotti L, et al. (2014) Genetic association study of QT interval highlights role for calcium signaling pathways in myocardial repolarization. Nature Genetics. 46: 826-36
et al. (2014) Genome-wide trans-ancestry meta-analysis provides insight into the genetic architecture of type 2 diabetes susceptibility. Nature Genetics. 46: 234-44
Anttila V, Winsvold BS, Gormley P, et al. (2013) Genome-wide meta-analysis identifies new susceptibility loci for migraine. Nature Genetics. 45: 912-7
Li G, Diogo D, Wu D, et al. (2013) Human genetics in rheumatoid arthritis guides a high-throughput drug screen of the CD40 signaling pathway. Plos Genetics. 9: e1003487
den Hoed M, Eijgelsheim M, Esko T, et al. (2013) Identification of heart rate-associated loci and their effects on cardiac conduction and rhythm disorders. Nature Genetics. 45: 621-31
Morris AP, Voight BF, Teslovich TM, et al. (2012) Large-scale association analysis provides insights into the genetic architecture and pathophysiology of type 2 diabetes. Nature Genetics. 44: 981-90
Talkowski ME, Rosenfeld JA, Blumenthal I, et al. (2012) Sequencing chromosomal abnormalities reveals neurodevelopmental loci that confer risk across diagnostic boundaries. Cell. 149: 525-37
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