Irena Levitan
Affiliations: | University of Pennsylvania, Philadelphia, PA, United States |
Area:
Biomedical Engineering, Cell BiologyGoogle:
"Irena Levitan"
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Publications
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| Beverley KM, Barbera N, Levitan I. (2024) Dual pattern of cholesterol-induced decoupling of residue-residue interactions of Kir2.2. Journal of Structural Biology. 216: 108091 |
| Beverley KM, Levitan I. (2024) Cholesterol regulation of mechanosensitive ion channels. Frontiers in Cell and Developmental Biology. 12: 1352259 |
| Fancher IS, Levitan I. (2023) Membrane Cholesterol Interactions with Proteins in Hypercholesterolemia-Induced Endothelial Dysfunction. Current Atherosclerosis Reports. 25: 535-541 |
| Barbera N, Granados ST, Vanoye CG, et al. (2022) Cholesterol-induced suppression of Kir2 channels is mediated by decoupling at the inter-subunit interfaces. Iscience. 25: 104329 |
| Rezende L, Couto NFD, Fernandes-Braga W, et al. (2022) OxLDL induces membrane structure rearrangement leading to biomechanics alteration and migration deficiency in macrophage. Biochimica Et Biophysica Acta. Biomembranes. 1864: 183951 |
| Levitan I. (2021) Evaluating membrane structure by Laurdan imaging: Disruption of lipid packing by oxidized lipids. Current Topics in Membranes. 88: 235-256 |
| Ahn SJ, Fancher IS, Granados ST, et al. (2021) Cholesterol-Induced Suppression of Endothelial Kir Channels Is a Driver of Impairment of Arteriolar Flow-Induced Vasodilation in Humans. Hypertension (Dallas, Tex. : 1979). HYPERTENSIONAHA12117 |
| Ayee MAA, Levitan I. (2021) Lipoprotein-Induced Increases in Cholesterol and 7-Ketocholesterol Result in Opposite Molecular-Scale Biophysical Effects on Membrane Structure. Frontiers in Cardiovascular Medicine. 8: 715932 |
| Srivastava N, Tauseef M, Amin R, et al. (2020) Noncanonical function of long myosin light chain kinase in increasing ER-PM junctions and augmentation of SOCE. Faseb Journal : Official Publication of the Federation of American Societies For Experimental Biology |
| Fancher IS, Le Master E, Ahn SJ, et al. (2020) Impairment of Flow-Sensitive Inwardly Rectifying K Channels via Disruption of Glycocalyx Mediates Obesity-Induced Endothelial Dysfunction. Arteriosclerosis, Thrombosis, and Vascular Biology. ATVBAHA120314935 |