Thomas H. Meek
Affiliations: | University of California, Riverside, Riverside, CA, United States |
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"Thomas Meek"
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Publications
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Dorfman MD, Monfeuga T, Melhorn SJ, et al. (2023) Central androgen action reverses hypothalamic astrogliosis and atherogenic risk factors induced by orchiectomy and high-fat diet feeding in male mice. American Journal of Physiology. Endocrinology and Metabolism |
Wone BWM, Yim WC, Schutz H, et al. (2018) Mitochondrial haplotypes are not associated with mice selectively bred for high voluntary wheel running. Mitochondrion |
Dorfman MD, Krull JE, Douglass JD, et al. (2017) Sex differences in microglial CX3CR1 signalling determine obesity susceptibility in mice. Nature Communications. 8: 14556 |
Acosta W, Meek TH, Schutz H, et al. (2016) Preference for Western diet coadapts in High Runner mice and affects voluntary exercise and spontaneous physical activity in a genotype-dependent manner. Behavioural Processes |
Malisch JL, deWolski K, Meek TH, et al. (2016) Acute Restraint Stress Alters Wheel-Running Behavior Immediately Following Stress and up to 20 Hours Later in House Mice. Physiological and Biochemical Zoology : Pbz. 89: 546-552 |
Morton GJ, Meek TH, Matsen ME, et al. (2015) Evidence against hypothalamic-pituitary-adrenal axis suppression in the antidiabetic action of leptin. The Journal of Clinical Investigation. 2015 |
Acosta W, Meek TH, Schutz H, et al. (2015) Effects of early-onset voluntary exercise on adult physical activity and associated phenotypes in mice. Physiology & Behavior. 149: 279-86 |
Meek TH, Dorfman MD, Matsen ME, et al. (2015) Evidence That in Uncontrolled Diabetes, Hyperglucagonemia Is Required for Ketosis but Not for Increased Hepatic Glucose Production or Hyperglycemia. Diabetes. 64: 2376-87 |
Meek TH, Matsen ME, Damian V, et al. (2014) Role of melanocortin signaling in neuroendocrine and metabolic actions of leptin in male rats with uncontrolled diabetes. Endocrinology. 155: 4157-67 |
Morton GJ, Meek TH, Schwartz MW. (2014) Neurobiology of food intake in health and disease. Nature Reviews. Neuroscience. 15: 367-78 |