Daniel P. Kane, Ph.D.
Affiliations: | Biology | Tufts University, Boston |
Area:
DNA repairGoogle:
"Daniel Kane"Parents
Sign in to add mentor| Mitch McVey | grad student | 2012 | Tufts | |
| (Multiple DNA polymerases act during homologous recombination repair in Drosophila melanogaster.) | ||||
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Publications
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| Xing X, Kane DP, Bulock CR, et al. (2019) A recurrent cancer-associated substitution in DNA polymerase ε produces a hyperactive enzyme. Nature Communications. 10: 374 |
| Barbari SR, Kane DP, Moore EA, et al. (2018) Functional Analysis of Cancer-Associated DNA Polymerase ε Variants in Saccharomyces cerevisiae. G3 (Bethesda, Md.) |
| Kane DP, Shcherbakova PV. (2014) A common cancer-associated DNA polymerase ε mutation causes an exceptionally strong mutator phenotype, indicating fidelity defects distinct from loss of proofreading. Cancer Research. 74: 1895-901 |
| Kane DP, Shusterman M, Rong Y, et al. (2012) Competition between replicative and translesion polymerases during homologous recombination repair in Drosophila. Plos Genetics. 8: e1002659 |
| Witsell A, Kane DP, McVey M. (2010) Super-sized deletions: improved transposon excision screens using a mus309 mutant background. Fly. 4: 137-40 |
| Witsell A, Kane DP, Rubin S, et al. (2009) Removal of the bloom syndrome DNA helicase extends the utility of imprecise transposon excision for making null mutations in Drosophila. Genetics. 183: 1187-93 |
| Conrad LJ, Bai L, Ahern K, et al. (2007) State II dissociation element formation following activator excision in maize. Genetics. 177: 737-47 |