Brendan J. Battersby, Ph.D.
Affiliations: | 2002 | McGill University, Montreal, QC, Canada |
Area:
GeneticsGoogle:
"Brendan Battersby"Parents
Sign in to add mentor| Eric A. Shoubridge | grad student | 2002 | McGill | |
| (Genetic basis for mtDNA segregation in a heteroplasmic mouse model.) | ||||
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Publications
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| Pratt SL, Zarate-Mendez M, Koludarova L, et al. (2025) Evaluating the feasibility of gene replacement strategies to treat MTRFR deficiency. Disease Models & Mechanisms. 18 |
| Teixeira J, Harju AM, Othman A, et al. (2024) Coenzyme Q improves mitochondrial and muscle dysfunction caused by CUG expanded repeats in Caenorhabditis elegans. Genetics |
| Hilander T, Awadhpersad R, Monteuuis G, et al. (2024) Supernumerary proteins of the human mitochondrial ribosomal small subunit are integral for assembly and translation. Iscience. 27: 110185 |
| Koludarova L, Battersby BJ. (2024) Mitochondrial protein synthesis quality control. Human Molecular Genetics |
| Gorski K, Jackson CB, Nyman TA, et al. (2023) Progressive mitochondrial dysfunction in cerebellar synaptosomes of cystatin B-deficient mice. Frontiers in Molecular Neuroscience. 16: 1175851 |
| Ng KY, Battersby BJ. (2023) Sucrose Gradient Analysis of Human Mitochondrial Ribosomes and RNA. Methods in Molecular Biology (Clifton, N.J.). 2661: 101-117 |
| Tomoda E, Nagao A, Shirai Y, et al. (2023) Restoration of mitochondrial function through activation of hypomodified tRNAs with pathogenic mutations associated with mitochondrial diseases. Nucleic Acids Research |
| Ng KY, Lutfullahoglu Bal G, Richter U, et al. (2022) Nonstop mRNAs generate a ground state of mitochondrial gene expression noise. Science Advances. 8: eabq5234 |
| Jett KA, Baker ZN, Hossain A, et al. (2022) Mitochondrial dysfunction reactivates α-fetoprotein expression that drives copper-dependent immunosuppression in mitochondrial disease models. The Journal of Clinical Investigation |
| Ng KY, Richter U, Jackson CB, et al. (2021) Translation of MT-ATP6 pathogenic variants reveals distinct regulatory consequences from the co-translational quality control of mitochondrial protein synthesis. Human Molecular Genetics |