Nicos Nicola
Affiliations: | The Walter and Eliza Hall Institute of Medical Research, Parkville, Victoria, Australia |
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Doggett K, Keating N, Dehkhoda F, et al. (2023) The SOCS1 KIR and SH2 domain are both required for suppression of cytokine signaling in vivo. Cytokine. 165: 156167 |
Tullett KM, Tan PS, Park HY, et al. (2020) RNF41 regulates the damage recognition receptor Clec9A and antigen cross-presentation in mouse dendritic cells. Elife. 9 |
Jackson JT, Shields BJ, Shi W, et al. (2017) Hhex Regulates Hematopoietic Stem Cell Self-Renewal and Stress Hematopoiesis via Repression of Cdkn2a. Stem Cells (Dayton, Ohio) |
Gough NM, Gough J, Metcalf D, et al. (2017) Pillars Article: Molecular Cloning of cDNA Encoding a Murine Haematopoietic Growth Regulator, Granulocyte-Macrophage Colony Stimulating Factor. Nature. 1984. 309: 763-767. Journal of Immunology (Baltimore, Md. : 1950). 198: 2522-2526 |
Ushiki T, Huntington ND, Glaser SP, et al. (2016) Rapid Inflammation in Mice Lacking Both SOCS1 and SOCS3 in Hematopoietic Cells. Plos One. 11: e0162111 |
Zhang JG, Farley A, Nicholson SE, et al. (2015) Correction for Zhang et al., The conserved SOCS box motif in suppressors of cytokine signaling binds to elongins B and C and may couple bound proteins to proteasomal degradation. Proceedings of the National Academy of Sciences of the United States of America. 112: E2979 |
Kedzierski L, Linossi EM, Kolesnik TB, et al. (2014) Suppressor of cytokine signaling 4 (SOCS4) protects against severe cytokine storm and enhances viral clearance during influenza infection. Plos Pathogens. 10: e1004134 |
Murphy JM, Czabotar PE, Hildebrand JM, et al. (2013) The pseudokinase MLKL mediates necroptosis via a molecular switch mechanism. Immunity. 39: 443-53 |
Croker BA, Kiu H, Pellegrini M, et al. (2012) IL-6 promotes acute and chronic inflammatory disease in the absence of SOCS3. Immunology and Cell Biology. 90: 124-9 |
Menkhorst E, Zhang JG, Sims NA, et al. (2011) Vaginally administered PEGylated LIF Antagonist blocked embryo implantation and eliminated non-target effects on bone in mice Plos One. 6 |