Yao Wei Lu

Affiliations: 
2028-2023 Cardiology Boston Children's Hospital & Harvard Medical School 
Google:
"Yao Lu"

Parents

Sign in to add mentor
Da-Zhi Wang post-doc 2018-2023 Boston Children's Hospital & Harvard Medical School
 (Heart development, RNA splicing)
BETA: Related publications

Publications

You can help our author matching system! If you notice any publications incorrectly attributed to this author, please sign in and mark matches as correct or incorrect.

Pan B, Hu D, Lu YW, et al. (2025) Trbp inhibits cardiac fibrosis through TGF-β pathway mediated crosstalk between cardiomyocytes and fibroblasts. Clinical Science (London, England : 1979)
de Oliveira Silva T, Lunardon G, Lino CA, et al. (2024) Senescent Cell Depletion Alleviates Obesity-related Metabolic and Cardiac Disorders. Molecular Metabolism. 102065
Wu T, Lu Y, Yu Y, et al. (2024) Long noncoding RNA AK144717 exacerbates pathological cardiac hypertrophy through modulating the cellular distribution of HMGB1 and subsequent DNA damage response. Cellular and Molecular Life Sciences : Cmls. 81: 432
Peng Q, Arulsamy K, Lu YW, et al. (2024) Novel Role of Endothelial CD45 in Regulating Endothelial-to-Mesenchymal Transition in Atherosclerosis. Biorxiv : the Preprint Server For Biology
Wang Y, Zhang M, Wang R, et al. (2024) Therapeutic Inhibition of Protects Against Cardiac Hypertrophy. Circulation Research
He X, Yang T, Lu YW, et al. (2024) The long non-coding RNA CARDINAL attenuates cardiac hypertrophy by modulating protein translation. The Journal of Clinical Investigation
Lu Y, Wang J, Yu Q. (2023) Next step of 'epileptic heart'. European Heart Journal
Gao F, Liang T, Lu YW, et al. (2023) Reduced Mitochondrial Protein Translation Promotes Cardiomyocyte Proliferation and Heart Regeneration. Circulation
Lu YW, Liang Z, Guo H, et al. (2023) PCBP1 regulates alternative splicing of AARS2 in congenital cardiomyopathy. Biorxiv : the Preprint Server For Biology
Gao F, Liang T, Lu YW, et al. (2023) A defect in mitochondrial protein translation influences mitonuclear communication in the heart. Nature Communications. 14: 1595
See more...