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Michael S. Killian, Ph.D.

Affiliations: 
2002 University of California, Los Angeles, Los Angeles, CA 
Area:
Epidemiology
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"Michael Killian"
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Roger Detels grad student 2002 UCLA
 (The antigen -specific T-cell response to HIV-1 infection.)

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Publications

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Killian MS, Teque F, Sudhagoni R. (2017) Analysis of the CD8 T cell anti-HIV activity in heterologous cell co-cultures reveals the benefit of multiple HLA class I matches. Immunogenetics. 70: 99-113
Killian MS, Fujimura SH, Sudhagoni RG. (2017) Brief Report: Increased Expression of the Type I Interferon Receptor on CD4+ T Lymphocytes in HIV-1-Infected Individuals. Journal of Acquired Immune Deficiency Syndromes (1999). 74: 473-478
Killian MS, Teque F, Walker RL, et al. (2013) CD8+ lymphocytes suppress human immunodeficiency virus 1 replication by secreting type i Interferons Journal of Interferon and Cytokine Research. 33: 632-645
Killian MS. (2012) Dual role of autophagy in HIV-1 replication and pathogenesis. Aids Research and Therapy. 9: 16
Vyas GN, Stoddart CA, Killian MS, et al. (2012) Derivation of non-infectious envelope proteins from virions isolated from plasma negative for HIV antibodies. Biologicals : Journal of the International Association of Biological Standardization. 40: 15-20
Killian MS, Levy JA. (2011) HIV/AIDS: 30 years of progress and future challenges. European Journal of Immunology. 41: 3401-11
Killian MS, Johnson C, Teque F, et al. (2011) Natural suppression of human immunodeficiency virus type 1 replication is mediated by transitional memory CD8+ T cells. Journal of Virology. 85: 1696-705
Killian MS, Roop J, Ng S, et al. (2009) CD8+ cell anti-HIV activity rapidly increases upon discontinuation of early antiretroviral therapy. Journal of Clinical Immunology. 29: 311-8
Killian MS, Norris PJ, Rawal BD, et al. (2006) The effects of early antiretroviral therapy and its discontinuation on the HIV-specific antibody response. Aids Research and Human Retroviruses. 22: 640-7
Killian MS, Fujimura SH, Hecht FM, et al. (2006) Similar changes in plasmacytoid dendritic cell and CD4 T-cell counts during primary HIV-1 infection and treatment. Aids (London, England). 20: 1247-52
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