David L. Sacks, Ph.D.
Affiliations: | Laboratory of Parasitic Diseases | National Institutes of Health, National Institute of Allergy and Infectious Diseases |
Area:
Immunology, Parasitology, Vector BiologyWebsite:
http://www.niaid.nih.gov/labsandresources/labs/aboutlabs/lpd/intracellularparasitebiologysection/Pages/sacks.aspxGoogle:
"David Sacks"Children
Sign in to add trainee| Emma Duge | research assistant | 2019-2020 | NIH (Neurotree) |
| Mary Ann McDowell | post-doc | 1997-2001 | NIAID, NIH |
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Publications
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| Lopes ME, Dos Santos LM, Sacks D, et al. (2021) Resistance Against Infection Depends on Microbiota-Guided Macrophage Activation. Frontiers in Immunology. 12: 730437 |
| Chaves MM, Lee SH, Kamenyeva O, et al. (2020) The role of dermis resident macrophages and their interaction with neutrophils in the early establishment of Leishmania major infection transmitted by sand fly bite. Plos Pathogens. 16: e1008674 |
| Singh OP, Hasker E, Boelaert M, et al. (2020) Xenodiagnosis to address key questions in visceral leishmaniasis control and elimination. Plos Neglected Tropical Diseases. 14: e0008363 |
| Singh OP, Syn G, Nylén S, et al. (2020) Anti-Interleukin-10 Unleashes Transcriptional Response to Leishmanial Antigens in Visceral Leishmaniasis Patients. The Journal of Infectious Diseases |
| Sacks DL. (2020) The Null Hypothesis of IFN-γ and Monocyte Function in Leishmaniasis. Cell Host & Microbe. 27: 683-684 |
| Louradour I, Ferreira TR, Ghosh K, et al. (2020) In Vitro Generation of Leishmania Hybrids. Cell Reports. 31: 107507 |
| Lewis MD, Paun A, Romano A, et al. (2020) Fatal progression of experimental visceral leishmaniasis is associated with intestinal parasitism and secondary infection by commensal bacteria, and is delayed by antibiotic prophylaxis. Plos Pathogens. 16: e1008456 |
| Lee SH, Chaves MM, Kamenyeva O, et al. (2020) M2-like, dermal macrophages are maintained via IL-4/CCL24-mediated cooperative interaction with eosinophils in cutaneous leishmaniasis. Science Immunology. 5 |
| Kumar R, Bunn PT, Singh SS, et al. (2020) Type I Interferons Suppress Anti-parasitic Immunity and Can Be Targeted to Improve Treatment of Visceral Leishmaniasis. Cell Reports. 30: 2512-2525.e9 |
| Louradour I, Ghosh K, Inbar E, et al. (2019) CRISPR/Cas9 Mutagenesis in Phlebotomus papatasi: the Immune Deficiency Pathway Impacts Vector Competence for Leishmania major. Mbio. 10 |