Gustavo Quirino

Immunology; Microbiology; Infectious Diseases

Joao Santana	research assistant	2013-2015	Ribeirao Preto Medical School
Russell E. Vance	grad student	2018-2018	HHMI, UC Berkeley
(Visiting Scholar at Russell Vance's lab)
Dario Zamboni	grad student	2015-2019	Ribeirao Preto Medical School

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Lima MHF, Sacramento LA, Quirino GFS, et al. (2017) Leishmania infantum Parasites Subvert the Host Inflammatory Response through the Adenosine A2A Receptor to Promote the Establishment of Infection. Frontiers in Immunology. 8: 815
Cunha LD, Silva ALN, Ribeiro JM, et al. (2017) AIM2 Engages Active but Unprocessed Caspase-1 to Induce Noncanonical Activation of the NLRP3 Inflammasome. Cell Reports. 20: 794-805
Franco LH, Fleuri AKA, Pellison NC, et al. (2017) Autophagy downstream of endosomal Toll-like receptor signaling in macrophages is a key mechanism for resistance to infection. The Journal of Biological Chemistry. 292: 13087-13096
Nascimento MS, Ferreira MD, Quirino GF, et al. (2016) NOD2-RIP2-mediated signalling contributes to shape adaptive immunity in visceral leishmaniasis. The Journal of Infectious Diseases
Davoli-Ferreira M, Fonseca DM, Mota CM, et al. (2016) Nucleotide-binding oligomerization domain-containing protein 2 prompts potent inflammatory stimuli during Neospora caninum infection. Scientific Reports. 6: 29289
Quirino GF, Nascimento MS, Davoli-Ferreira M, et al. (2016) Interleukin-27 Mediates Susceptibility to Visceral Leishmaniasis by Suppressing the IL-17-Neutrophil Response. Infection and Immunity