Arjun Rustagi, M.D./Ph.D.
Affiliations: | 2015- | Medicine | Stanford University Medical School, Palo Alto, CA, United States |
Area:
virology, immunology, computational biology, lung biology, innate immunityWebsite:
https://med.stanford.edu/profiles/arjun-rustagiGoogle:
"Arjun Rustagi"Parents
Sign in to add mentor| Michael Gale Jr. | grad student | 2009-2013 | University of Washington (Microtree) |
| Catherine A Blish | post-doc | 2018-2021 | Stanford |
BETA: Related publications
See more...
Publications
|
You can help our author matching system! If you notice any publications incorrectly attributed to this author, please sign in and mark matches as correct or incorrect. |
| Lee MJ, Leong MW, Rustagi A, et al. (2022) SARS-CoV-2 escapes direct NK cell killing through Nsp1-mediated downregulation of ligands for NKG2D. Cell Reports. 41: 111892 |
| Martínez-Colón GJ, Ratnasiri K, Chen H, et al. (2022) SARS-CoV-2 infection drives an inflammatory response in human adipose tissue through infection of adipocytes and macrophages. Science Translational Medicine. eabm9151 |
| Richards CM, Jabs S, Qiao W, et al. (2022) The human disease gene LYSET is essential for lysosomal enzyme transport and viral infection. Science (New York, N.Y.). eabn5648 |
| Hagey RJ, Elazar M, Pham EA, et al. (2022) Programmable antivirals targeting critical conserved viral RNA secondary structures from influenza A virus and SARS-CoV-2. Nature Medicine |
| Zhu C, Lee JY, Woo JZ, et al. (2022) An intranasal ASO therapeutic targeting SARS-CoV-2. Nature Communications. 13: 4503 |
| Biering SB, Sarnik SA, Wang E, et al. (2022) Genome-wide bidirectional CRISPR screens identify mucins as host factors modulating SARS-CoV-2 infection. Nature Genetics |
| Zeng L, Liu Y, Nguyenla XH, et al. (2022) Broad-spectrum CRISPR-mediated inhibition of SARS-CoV-2 variants and endemic coronaviruses in vitro. Nature Communications. 13: 2766 |
| Yen M, Ren J, Liu Q, et al. (2022) Facile discovery of surrogate cytokine agonists. Cell |
| Carroll T, Fox D, van Doremalen N, et al. (2022) The B.1.427/1.429 (epsilon) SARS-CoV-2 variants are more virulent than ancestral B.1 (614G) in Syrian hamsters. Plos Pathogens. 18: e1009914 |
| Carroll T, Fox D, van Doremalen N, et al. (2021) The B.1.427/1.429 (epsilon) SARS-CoV-2 variants are more virulent than ancestral B.1 (614G) in Syrian hamsters. Biorxiv : the Preprint Server For Biology |