Jimo Borjigin
Affiliations: | Molecular and Integrative Physiology | University of Michigan, Ann Arbor, Ann Arbor, MI |
Area:
Circadian RhythmsGoogle:
"Jimo Borjigin"Cross-listing: Neurotree - Chemistry Tree
Children
Sign in to add trainee| L. Samantha S. Zhang | grad student | 2008-2011 | University of Michigan (Neurotree) |
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Publications
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| Dean JG, Liu T, Huff S, et al. (2019) Biosynthesis and Extracellular Concentrations of N,N-dimethyltryptamine (DMT) in Mammalian Brain. Scientific Reports. 9: 9333 |
| Tian F, Liu T, Xu G, et al. (2019) Surge of Corticocardiac Coupling in SHRSP Rats Exposed to Forebrain Cerebral Ischemia. Journal of Neurophysiology |
| Xu G, Dean J, Liu T, et al. (2018) Chronic circadian advance shifts abolish melatonin secretion for days in rats. Neurobiology of Sleep and Circadian Rhythms. 5: 78-83 |
| Tian F, Liu T, Xu G, et al. (2018) Adrenergic Blockade Bi-directionally and Asymmetrically Alters Functional Brain-Heart Communication and Prolongs Electrical Activities of the Brain and Heart during Asphyxic Cardiac Arrest. Frontiers in Physiology. 9: 99 |
| Wadas B, Borjigin J, Huang Z, et al. (2016) Degradation of Serotonin N-Acetyltransferase, a Circadian Regulator, by the N-end Rule Pathway. The Journal of Biological Chemistry |
| Li D, Mabrouk OS, Liu T, et al. (2015) Asphyxia-activated corticocardiac signaling accelerates onset of cardiac arrest. Proceedings of the National Academy of Sciences of the United States of America. 112: E2073-82 |
| Borjigin J, Zhang LS, Calinescu AA. (2012) Circadian regulation of pineal gland rhythmicity. Molecular and Cellular Endocrinology. 349: 13-9 |
| Huang Z, Liu T, Borjigin J. (2010) N-terminal residues regulate proteasomal degradation of AANAT. Journal of Pineal Research. 48: 290-6 |
| Chattoraj A, Liu T, Zhang LS, et al. (2009) Melatonin formation in mammals: in vivo perspectives. Reviews in Endocrine & Metabolic Disorders. 10: 237-43 |
| Schwartz MD, Wotus C, Liu T, et al. (2009) Dissociation of circadian and light inhibition of melatonin release through forced desynchronization in the rat. Proceedings of the National Academy of Sciences of the United States of America. 106: 17540-5 |