Robert L. Furler, Ph.D.
Affiliations: | 2010 | University of California, Los Angeles, Los Angeles, CA |
Area:
Immunology, Molecular Biology, Human DevelopmentGoogle:
"Robert Furler"Parents
Sign in to add mentor| Christel H. Uittenbogaart | grad student | 2010 | UCLA | |
| (Transcriptional Regulation of the Human TGF-beta1 Gene by the GLI Transcription Factors.) | ||||
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Publications
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| Duarte RRR, Pain O, Furler RL, et al. (2022) Transcriptome-wide association study of HIV-1 acquisition identifies as a susceptibility gene. Iscience. 25: 104854 |
| Furler RL, Nixon DF. (2021) HIV-1 Exploits Transitions Between CD4 T Cell Migration and Activation. Aids Research and Human Retroviruses |
| Premeaux TA, Mediouni S, Leda A, et al. (2021) Next-Generation Human Cerebral Organoids as Powerful Tools To Advance NeuroHIV Research. Mbio. e0068021 |
| Sarabia I, Novis CL, Macedo AB, et al. (2021) Activation of the Anti-Oxidative Stress Response Reactivates Latent HIV-1 Through the Mitochondrial Antiviral Signaling Protein Isoform MiniMAVS. Frontiers in Immunology. 12: 682182 |
| Curty G, Beckerle GA, Iñiguez LP, et al. (2020) Human Endogenous Retrovirus Expression Is Upregulated in the Breast Cancer Microenvironment of HIV Infected Women: A Pilot Study. Frontiers in Oncology. 10: 553983 |
| Ali A, Furler RL, Pedroza-Martins L, et al. (2019) A Novel HIV-1 Nef Mutation in a Primary Pediatric Isolate Impairs MHC-Class I Downregulation and Cytopathicity. Aids Research and Human Retroviruses |
| Furler RL, Newcombe KL, Del Rio P, et al. (2019) Histoarchitectural Deterioration of Lymphoid Tissues in HIV-1 Infection and in Aging. Aids Research and Human Retroviruses |
| Furler RL, Ali A, Yang OO, et al. (2019) Nef-induced differential gene expression in primary CD4+ T cells following infection with HIV-1 isolates. Virus Genes |
| Furler RL, Nixon DF. (2019) The Intimate Relationship Between CD4+ T Cell Morphology and HIV-1 Infection. Aids Research and Human Retroviruses |
| Furler RL, Nixon DF, Brantner CA, et al. (2018) TGF-β Sustains Tumor Progression through Biochemical and Mechanical Signal Transduction. Cancers. 10 |