Jobert G. Barin, Ph.D.
Affiliations: | 2010 | Johns Hopkins University, Baltimore, MD |
Area:
Immunology, PathologyGoogle:
"Jobert Barin"Parents
Sign in to add mentor| Noel R. Rose | grad student | 2010 | Johns Hopkins | |
| (Cytokines, inflammation, and the participation of macrophages in autoimmune heart disease.) | ||||
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Publications
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| Hou X, Chen G, Bracamonte-Baran W, et al. (2019) The Cardiac Microenvironment Instructs Divergent Monocyte Fates and Functions in Myocarditis. Cell Reports. 28: 172-189.e7 |
| Barin JG, Talor MV, Diny NL, et al. (2017) Regulation of autoimmune myocarditis by host responses to the microbiome. Experimental and Molecular Pathology |
| Fontes JA, Barin JG, Talor MV, et al. (2017) Complete Freund's adjuvant induces experimental autoimmune myocarditis by enhancing IL-6 production during initiation of the immune response. Immunity, Inflammation and Disease. 5: 163-176 |
| Diny NL, Baldeviano GC, Talor MV, et al. (2017) Eosinophil-derived IL-4 drives progression of myocarditis to inflammatory dilated cardiomyopathy. The Journal of Experimental Medicine |
| Diny NL, Hou X, Barin JG, et al. (2016) Macrophages and cardiac fibroblasts are the main producers of eotaxins and regulate eosinophil trafficking to the heart. European Journal of Immunology |
| Selmi C, Barin JG, Rose NR. (2016) Current trends in autoimmunity and the nervous system. Journal of Autoimmunity |
| Barin JG, Talor MV, Schaub JA, et al. (2016) Collaborative Interferon-γ and Interleukin-17 Signaling Protects the Oral Mucosa from Staphylococcus aureus. The American Journal of Pathology |
| Wu L, Diny NL, Ong S, et al. (2015) Pathogenic IL-23 signaling is required to initiate GM-CSF-driven autoimmune myocarditis in mice. European Journal of Immunology |
| Barin JG, Tobias LD, Peterson DA. (2015) The microbiome and autoimmune disease: Report from a Noel R. Rose Colloquium. Clinical Immunology (Orlando, Fla.). 159: 183-8 |
| Ong S, Ligons DL, Barin JG, et al. (2015) Natural killer cells limit cardiac inflammation and fibrosis by halting eosinophil infiltration. The American Journal of Pathology. 185: 847-61 |