Christopher L. Case, Ph.D.
Affiliations: | 2011 | Yale University, New Haven, CT |
Area:
Immunology, Microbiology Biology, PathologyGoogle:
"Christopher Case"Parents
Sign in to add mentor| Craig R. Roy | grad student | 2011 | Yale | |
| (Regulation of Caspase-1-associated Activities in Macrophages During Legionella pneumophila Infection.) | ||||
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Publications
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| Case CL, Kohler LJ, Lima JB, et al. (2013) Caspase-11 stimulates rapid flagellin-independent pyroptosis in response to Legionella pneumophila. Proceedings of the National Academy of Sciences of the United States of America. 110: 1851-6 |
| Case CL, Roy CR. (2013) Analyzing caspase-1 activation during Legionella pneumophila infection in macrophages. Methods in Molecular Biology (Clifton, N.J.). 954: 479-91 |
| Case CL. (2011) Regulating caspase-1 during infection: roles of NLRs, AIM2, and ASC. The Yale Journal of Biology and Medicine. 84: 333-43 |
| Case CL, Roy CR. (2011) Asc modulates the function of NLRC4 in response to infection of macrophages by Legionella pneumophila. Mbio. 2 |
| Nogueira CV, Lindsten T, Jamieson AM, et al. (2009) Rapid pathogen-induced apoptosis: a mechanism used by dendritic cells to limit intracellular replication of Legionella pneumophila. Plos Pathogens. 5: e1000478 |
| Case CL, Shin S, Roy CR. (2009) Asc and Ipaf Inflammasomes direct distinct pathways for caspase-1 activation in response to Legionella pneumophila. Infection and Immunity. 77: 1981-91 |
| Shin S, Case CL, Archer KA, et al. (2008) Type IV secretion-dependent activation of host MAP kinases induces an increased proinflammatory cytokine response to Legionella pneumophila. Plos Pathogens. 4: e1000220 |