Jeffrey T. McNamara, Ph.D.
Affiliations: | 2011 | University of Connecticut, Storrs, CT, United States |
Area:
Microbiology Biology, Histology Biology, ImmunologyGoogle:
"Jeffrey McNamara"Parents
Sign in to add mentorRoger Thrall | grad student | 2011 | University of Connecticut | |
(Phenotypic and Functional Plasticity in the Endogenous CD8+ T cell Response Correlates with the Establishment and Resolution of Allergic Airway Disease.) |
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Publications
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McNamara JT, Huntington KE, Borys S, et al. (2021) SHP-2 deletion in CD4Cre expressing chondrocyte precursors leads to tumor development with wrist tropism. Scientific Reports. 11: 20006 |
Andemariam B, Adami AJ, Singh A, et al. (2015) The sickle cell mouse lung: proinflammatory and primed for allergic inflammation. Translational Research : the Journal of Laboratory and Clinical Medicine. 166: 254-68 |
Valenzuela NM, McNamara JT, Reed EF. (2014) Antibody-mediated graft injury: complement-dependent and complement-independent mechanisms. Current Opinion in Organ Transplantation. 19: 33-40 |
Secor ER, Szczepanek SM, Castater CA, et al. (2013) Bromelain Inhibits Allergic Sensitization and Murine Asthma via Modulation of Dendritic Cells. Evidence-Based Complementary and Alternative Medicine : Ecam. 2013: 702196 |
Szczepanek SM, McNamara JT, Secor ER, et al. (2012) Splenic morphological changes are accompanied by altered baseline immunity in a mouse model of sickle-cell disease. The American Journal of Pathology. 181: 1725-34 |
Natarajan P, Singh A, McNamara JT, et al. (2012) Regulatory B cells from hilar lymph nodes of tolerant mice in a murine model of allergic airway disease are CD5+, express TGF-β, and co-localize with CD4+Foxp3+ T cells. Mucosal Immunology. 5: 691-701 |
McNamara JT, Schramm CM, Singh A, et al. (2012) Phenotypic changes to the endogenous antigen-specific CD8+ T cell response correlates with the development and resolution of allergic airway disease. The American Journal of Pathology. 180: 1991-2000 |
Secor ER, Singh A, Guernsey LA, et al. (2009) Bromelain treatment reduces CD25 expression on activated CD4+ T cells in vitro. International Immunopharmacology. 9: 340-6 |