Samuel Boateng

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1999-2005 School of Biological Sciences University of Reading, UK, Earley, England, United Kingdom 
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"Samuel Boateng"
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Publications

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Pitoulis FG, Hasan W, Papadaki M, et al. (2020) Intact myocardial preparations reveal intrinsic transmural heterogeneity in cardiac mechanics. Journal of Molecular and Cellular Cardiology
Pierrat OA, Paudyal A, Woodruff J, et al. (2017) The exon junction complex senses energetic stress and regulates contractility and cell architecture in cardiac myocytes. Bioscience Reports. 37
Atkins J, Makewita L, Gargaro M, et al. (2017) Investigation of protein mutations associated with heart failure using bioinformatic tools F1000research. 6
Paudyal A, Dewan S, Ikie C, et al. (2016) Nuclear accumulation of myocyte muscle LIM protein is regulated by heme oxygenase 1 and correlates with cardiac function in the transition to failure. The Journal of Physiology
Paudyal A, Dewan S, Tombe PD, et al. (2014) Increased Passive And Contractile Stress Within The Myofilaments Contribute To The Loss Of Mechanosensing In The Heart During The Progression To Heart Failure Heart. 100
Hooper CL, Paudyal A, Dash PR, et al. (2013) Modulation of stretch-induced myocyte remodeling and gene expression by nitric oxide: a novel role for lipoma preferred partner in myofibrillogenesis. American Journal of Physiology. Heart and Circulatory Physiology. 304: H1302-13
Hooper CL, Dash PR, Boateng SY. (2012) Lipoma preferred partner is a mechanosensitive protein regulated by nitric oxide in the heart. Febs Open Bio. 2: 135-44
Boateng SY, Senyo SE, Qi L, et al. (2009) Myocyte remodeling in response to hypertrophic stimuli requires nucleocytoplasmic shuttling of muscle LIM protein. Journal of Molecular and Cellular Cardiology. 47: 426-35
Boateng SY, Senyo SE, Qi L, et al. (2008) Nucleocytoplasmic shuttling of muscle LIM protein is required for myocyte remodelling and adaptation to hypertrophic stimuli Journal of Molecular and Cellular Cardiology. 44: 792
Boateng SY, Belin RJ, Geenen DL, et al. (2007) Cardiac dysfunction and heart failure are associated with abnormalities in the subcellular distribution and amounts of oligomeric muscle LIM protein. American Journal of Physiology. Heart and Circulatory Physiology. 292: H259-69
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