Anna Huppler
Affiliations: | 2011-2014 | University of Pittsburgh, Pittsburgh, PA, United States |
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"Anna Huppler"
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| Jondle CN, Johnson KE, Aurubin C, et al. (2021) Gammaherpesvirus Usurps Host IL-17 Signaling To Support the Establishment of Chronic Infection. Mbio. 12 |
| Peters BM, Coleman BM, Willems HME, et al. (2019) The IL-17R/IL-22R signaling axis is dispensable for vulvovaginal candidiasis regardless of estrogen status. The Journal of Infectious Diseases |
| Claeys TA, Rosas Mejia O, Marshall S, et al. (2019) TH cells' capacity for TH1 and TH17 differentiation is attenuated in children with nontuberculous mycobacteria infection. The Journal of Infectious Diseases |
| Verma AH, Richardson JP, Zhou C, et al. (2017) Oral epithelial cells orchestrate innate type 17 responses to Candida albicans through the virulence factor candidalysin. Science Immunology. 2 |
| Huppler AR, Gaffen SL. (2016) Editorial: Fake it 'til you make it: mast cells acquire IL-17 exogenously. Journal of Leukocyte Biology. 100: 445-6 |
| Huppler AR, Verma AH, Conti HR, et al. (2015) Neutrophils Do Not Express IL-17A in the Context of Acute Oropharyngeal Candidiasis. Pathogens (Basel, Switzerland). 4: 559-72 |
| Huppler AR, Whibley N, Woolford CA, et al. (2015) A Candida albicans strain expressing mammalian IL-17A results in early control of fungal growth during disseminated infection. Infection and Immunity |
| Huppler AR, Conti HR, Hernández-Santos N, et al. (2015) Correction: Role of Neutrophils in IL-17-Dependent Immunity to Mucosal Candidiasis. Journal of Immunology (Baltimore, Md. : 1950). 194: 1382 |
| Conti HR, Peterson AC, Brane L, et al. (2014) Oral-resident natural Th17 cells and γδ T cells control opportunistic Candida albicans infections. The Journal of Experimental Medicine. 211: 2075-84 |
| Ramani K, Pawaria S, Maers K, et al. (2014) An essential role of interleukin-17 receptor signaling in the development of autoimmune glomerulonephritis. Journal of Leukocyte Biology. 96: 463-72 |