Akash Verma
Affiliations: | 2014-2019 | University of Pittsburgh, Pittsburgh, PA, United States |
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"Akash Verma"
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| Peters BM, Coleman BM, Willems HME, et al. (2019) The IL-17R/IL-22R signaling axis is dispensable for vulvovaginal candidiasis regardless of estrogen status. The Journal of Infectious Diseases |
| Verma AH, Gaffen SL. (2019) Dermatophyte Immune Memory Is Only Skin-Deep. The Journal of Investigative Dermatology. 139: 517-519 |
| Shen F, Verma AH, Volk A, et al. (2019) Combined Blockade of TNF-α and IL-17A Alleviates Progression of Collagen-Induced Arthritis without Causing Serious Infections in Mice. Journal of Immunology (Baltimore, Md. : 1950) |
| Verma AH, Zafar H, Ponde NO, et al. (2018) IL-36 and IL-1/IL-17 Drive Immunity to Oral Candidiasis via Parallel Mechanisms. Journal of Immunology (Baltimore, Md. : 1950) |
| Ramani K, Jawale CV, Verma AH, et al. (2018) Unexpected kidney-restricted role for IL-17 receptor signaling in defense against systemic Candida albicans infection. Jci Insight. 3 |
| Richardson JP, Mogavero S, Moyes DL, et al. (2018) Processing of Candida albicans Ece1p Is Critical for Candidalysin Maturation and Fungal Virulence. Mbio. 9 |
| Verma A, Gaffen SL, Swidergall M. (2017) Innate Immunity to Mucosal Candida Infections. Journal of Fungi (Basel, Switzerland). 3 |
| Verma AH, Richardson JP, Zhou C, et al. (2017) Oral epithelial cells orchestrate innate type 17 responses to Candida albicans through the virulence factor candidalysin. Science Immunology. 2 |
| Conti HR, Bruno VM, Childs EE, et al. (2016) IL-17 Receptor Signaling in Oral Epithelial Cells Is Critical for Protection against Oropharyngeal Candidiasis. Cell Host & Microbe. 20: 606-617 |
| Monin L, Gudjonsson JE, Childs EE, et al. (2016) MCPIP1/Regnase-1 Restricts IL-17A- and IL-17C-Dependent Skin Inflammation. Journal of Immunology (Baltimore, Md. : 1950) |