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| Ferrari M, Rawal CC, Lodovichi S, et al. (2020) Rad9/53BP1 promotes DNA repair via crossover recombination by limiting the Sgs1 and Mph1 helicases. Nature Communications. 11: 3181 |
| Dibitetto D, La Monica M, Ferrari M, et al. (2018) Formation and nucleolytic processing of Cas9-induced DNA breaks in human cells quantified by droplet digital PCR. Dna Repair. 68: 68-74 |
| Ferrari M, Twayana S, Marini F, et al. (2018) A qPCR-Based Protocol to Quantify DSB Resection. Methods in Molecular Biology (Clifton, N.J.). 1672: 119-129 |
| Rawal CC, Riccardo S, Pesenti C, et al. (2016) Reduced kinase activity of polo kinase Cdc5 affects chromosome stability and DNA damage response in S. cerevisiae. Cell Cycle (Georgetown, Tex.). 15: 2906-2919 |
| Dibitetto D, Ferrari M, Rawal CC, et al. (2015) Slx4 and Rtt107 control checkpoint signalling and DNA resection at double-strand breaks. Nucleic Acids Research |
| Ferrari M, Dibitetto D, De Gregorio G, et al. (2015) Functional interplay between the 53BP1-ortholog Rad9 and the Mre11 complex regulates resection, end-tethering and repair of a double-strand break. Plos Genetics. 11: e1004928 |
| Ferrari M, Nachimuthu BT, Donnianni RA, et al. (2013) Tid1/Rdh54 translocase is phosphorylated through a Mec1- and Rad53-dependent manner in the presence of DSB lesions in budding yeast. Dna Repair. 12: 347-55 |
| Rossio V, Galati E, Ferrari M, et al. (2010) The RSC chromatin-remodeling complex influences mitotic exit and adaptation to the spindle assembly checkpoint by controlling the Cdc14 phosphatase. The Journal of Cell Biology. 191: 981-97 |
| Donnianni RA, Ferrari M, Lazzaro F, et al. (2010) Elevated levels of the polo kinase Cdc5 override the Mec1/ATR checkpoint in budding yeast by acting at different steps of the signaling pathway. Plos Genetics. 6: e1000763 |