Marcello D'Amelio
Affiliations: | IRCCS Santa Lucia Foundation, Rome, Italy, Roma, Lazio, Italy |
Area:
NeurodegenerationGoogle:
"Marcello D'Amelio"Cross-listing: Neurotree
Parents
Sign in to add mentorFrancesco Cecconi | post-doc | 2005- | IRCCS Santa Lucia Foundation, Rome, Italy (Neurotree) |
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Publications
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Pignataro A, Krashia P, De Introna M, et al. (2023) Chemogenetic rectification of the inhibitory tone onto hippocampal neurons reverts autistic-like traits and normalizes local expression of estrogen receptors in the Ambra1+/- mouse model of female autism. Translational Psychiatry. 13: 63 |
Klionsky DJ, Abdel-Aziz AK, Abdelfatah S, et al. (2021) Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition). Autophagy. 1-382 |
Nobili A, Krashia P, D'Amelio M. (2020) Cisd2: A promising new target in Alzheimer's disease. The Journal of Pathology |
Borreca A, Valeri F, De Luca M, et al. (2020) Transient upregulation of translational efficiency in prodromal and early symptomatic Tg2576 mice contributes to Aβ pathology. Neurobiology of Disease. 104787 |
Becher J, Simula L, Volpe E, et al. (2018) AMBRA1 Controls Regulatory T-Cell Differentiation and Homeostasis Upstream of the FOXO3-FOXP3 Axis. Developmental Cell. 47: 592-607.e6 |
Cordella A, Krashia P, Nobili A, et al. (2018) Dopamine loss alters the hippocampus-nucleus accumbens synaptic transmission in the Tg2576 mouse model of Alzheimer's disease. Neurobiology of Disease |
D'Amelio M, Serra L, Bozzali M. (2018) Ventral Tegmental Area in Prodromal Alzheimer's Disease: Bridging the Gap between Mice and Humans. Journal of Alzheimer's Disease : Jad. 63: 181-183 |
Nobili A, Krashia P, Cordella A, et al. (2018) Ambra1 Shapes Hippocampal Inhibition/Excitation Balance: Role in Neurodevelopmental Disorders. Molecular Neurobiology |
Landrock KK, Sullivan P, Martini-Stoica H, et al. (2017) Pleiotropic Neuropathological and Biochemical Alterations Associated with Myo5a Mutation in a Rat Model. Brain Research |
Nobili A, Latagliata EC, Viscomi MT, et al. (2017) Dopamine neuronal loss contributes to memory and reward dysfunction in a model of Alzheimer's disease. Nature Communications. 8: 14727 |