Matt Kelley, PhD
Affiliations: | 2013-2018 | Neuroscience | Tufts University, Boston |
2018-2020 | Neuroscience | Amgen Inc., Thousand Oaks, CA, United States | |
2020-2021 | Voyager Therapeutics | ||
2021-2022 | Pfizer |
Area:
EpilepsyGoogle:
"Matt Kelley"Cross-listing: Neurotree
Parents
Sign in to add mentorIstvan Mody | research assistant | 2011-2013 | UCLA (Neurotree) |
Stephen J. Moss | grad student | 2014-2018 | Tufts (Neurotree) |
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Publications
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Kelley MR, Cardarelli RA, Smalley JL, et al. (2018) Locally Reducing KCC2 Activity in the Hippocampus is Sufficient to Induce Temporal Lobe Epilepsy. Ebiomedicine |
Moore YE, Kelley MR, Brandon NJ, et al. (2017) Seizing Control of KCC2: A New Therapeutic Target for Epilepsy. Trends in Neurosciences |
Cairns DM, Chwalek K, Moore YE, et al. (2016) Expandable and Rapidly Differentiating Human Induced Neural Stem Cell Lines for Multiple Tissue Engineering Applications. Stem Cell Reports |
Kelley MR, Deeb TZ, Brandon NJ, et al. (2016) Compromising KCC2 transporter activity enhances the development of continuous seizure activity. Neuropharmacology |
Sivakumaran S, Cardarelli RA, Maguire J, et al. (2015) Selective Inhibition of KCC2 Leads to Hyperexcitability and Epileptiform Discharges in Hippocampal Slices and In Vivo. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 35: 8291-6 |
Silayeva L, Deeb TZ, Hines RM, et al. (2015) KCC2 activity is critical in limiting the onset and severity of status epilepticus. Proceedings of the National Academy of Sciences of the United States of America. 112: 3523-8 |