Edward D. Hall, Ph.D.
Affiliations: | Anatomy & Neurobiology | University of Kentucky, Lexington, KY |
Area:
traumatic brain injury, spinal cord injury, secondary injuryWebsite:
https://neurobiology.med.uky.edu/users/edhallGoogle:
"Edward Hall"Children
Sign in to add trainee| Darren Miller | grad student | 2008- | University of Kentucky |
| Jacob Dunkerson | grad student | 2017- | University of Kentucky |
| Ying Deng-Bryant | grad student | 2007 | University of Kentucky |
| Lamin H. Mbye | grad student | 2008 | University of Kentucky |
| Yiqin Xiong-Smith | grad student | 2008 | University of Kentucky |
| Ayman G. Mustafa | grad student | 2010 | University of Kentucky |
| John E. Cebak | grad student | 2015 | University of Kentucky |
Collaborators
Sign in to add collaborator| Indrapal N. Singh | collaborator | 2004- | University of Kentucky (Neurotree) |
| Douglas K. Anderson | collaborator | 1982-1997 | University of Florida Dept of Neuroscience (Neurotree) |
BETA: Related publications
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Publications
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| LaPlaca MC, Huie JR, Alam HB, et al. (2020) Preclinical Common Data Elements for Traumatic Brain Injury Research: Progress and Use Cases. Journal of Neurotrauma |
| Hill RL, Singh IN, Wang JA, et al. (2020) Protective effects of phenelzine administration on synaptic and non-synaptic cortical mitochondrial function and lipid peroxidation-mediated oxidative damage following TBI in young adult male rats. Experimental Neurology. 113322 |
| Hill RL, Singh IN, Brelsfoard J, et al. (2020) Pharmacological inhibition of lipid peroxidative damage by the 21-aminosteroid U-74389G improves cortical mitochondrial function following traumatic brain injury in young adult male rats. Neuropharmacology. 108023 |
| Hill RL, Singh IN, Wang J, et al. (2018) THE EFFECTS OF PHENELZINE ADMINISTRATION OF MITOCHONDRIAL FUNCTION, CALCIUM HANDLING AND CYTOSKELETAL DEGRADATION FOLLOWING EXPERIMENTAL TRAUMATIC BRAIN INJURY. Journal of Neurotrauma |
| Hall ED, Wang JA, Miller DM, et al. (2018) Newer Pharmacological Approaches for Antioxidant Neuroprotection in Traumatic Brain Injury. Neuropharmacology |
| Hill RL, Kulbe JR, Singh IN, et al. (2018) Synaptic mitochondria are more susceptible to traumatic brain injury-induced oxidative damage and respiratory dysfunction than non-synaptic mitochondria. Neuroscience |
| DeWitt D, Hawkins BE, Dixon CE, et al. (2018) Preclinical Testing of Therapies for Traumatic Brain Injury. Journal of Neurotrauma |
| Kulbe JR, Singh IN, Wang J, et al. (2018) CONTINUOUS INFUSION OF PHENELZINE, CYCLOSPORINE A OR THE COMBINATION: EVALUATION OF MITOCHONDRIAL BIOENERGETICS, OXIDATIVE DAMAGE AND CYTOSKELETAL DEGRADATION FOLLOWING SEVERE CONTROLLED CORTICAL IMPACT TRAUMATIC BRAIN INJURY IN RATS. Journal of Neurotrauma |
| Springer JE, Visavadiya N, Sullivan PG, et al. (2017) Post-injury treatment with NIM811 promotes recovery of function in adult female rats following spinal cord contusion: A dose-response study. Journal of Neurotrauma |
| Vemuganti R, Hall ED. (2017) Cellular and molecular mechanisms of neuroprotection and plasticity after traumatic brain injury. Neurochemistry International |