Earl R. McDonald, Ph.D.
Affiliations: | 2003 | University of Pennsylvania, Philadelphia, PA, United States |
Area:
Molecular BiologyGoogle:
"Earl McDonald"Mean distance: (not calculated yet)
Parents
Sign in to add mentor| Wafik S. El-Deiry | grad student | 2003 | Penn | |
| (Determinants of killer /DR5 signaling and death ligand sensitivity.) | ||||
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| Mavrakis KJ, McDonald ER, Schlabach MR, et al. (2016) Disordered methionine metabolism in MTAP/CDKN2A deleted cancers leads to dependence on PRMT5. Science (New York, N.Y.) |
| Yang W, Rozan LM, McDonald ER, et al. (2007) CARPs are ubiquitin ligases that promote MDM2-independent p53 and phospho-p53ser20 degradation. The Journal of Biological Chemistry. 282: 3273-81 |
| Tibbetts MD, Shiozaki EN, Gu L, et al. (2004) Crystal structure of a FYVE-type zinc finger domain from the caspase regulator CARP2. Structure (London, England : 1993). 12: 2257-63 |
| Jin Z, McDonald ER, Dicker DT, et al. (2004) Deficient tumor necrosis factor-related apoptosis-inducing ligand (TRAIL) death receptor transport to the cell surface in human colon cancer cells selected for resistance to TRAIL-induced apoptosis. The Journal of Biological Chemistry. 279: 35829-39 |
| McDonald ER, El-Deiry WS. (2004) Suppression of caspase-8- and -10-associated RING proteins results in sensitization to death ligands and inhibition of tumor cell growth. Proceedings of the National Academy of Sciences of the United States of America. 101: 6170-5 |
| Corn PG, McDonald ER, Herman JG, et al. (2003) Tat-binding protein-1, a component of the 26S proteasome, contributes to the E3 ubiquitin ligase function of the von Hippel-Lindau protein. Nature Genetics. 35: 229-37 |
| McDonald ER, El-Deiry WS. (2003) Yeast two-hybrid screening as a means of deciphering tumor suppressor pathways. Methods in Molecular Biology (Clifton, N.J.). 223: 173-85 |
| McDonald ER, El-Deiry WS. (2001) Checkpoint genes in cancer. Annals of Medicine. 33: 113-22 |
| McDonald ER, Chui PC, Martelli PF, et al. (2001) Death domain mutagenesis of KILLER/DR5 reveals residues critical for apoptotic signaling. The Journal of Biological Chemistry. 276: 14939-45 |
| Meng RD, McDonald ER, Sheikh MS, et al. (2000) The TRAIL Decoy Receptor TRUNDD (DcR2, TRAIL-R4) Is Induced by Adenovirus-p53 Overexpression and Can Delay TRAIL-, p53-, and KILLER/DR5-Dependent Colon Cancer Apoptosis Molecular Therapy. 1: 130-144 |