Ronen Sumagin, Ph.D.
Affiliations: | 2008 | University of Rochester, Rochester, NY |
Area:
Biomedical Engineering, Physiology BiologyGoogle:
"Ronen Sumagin"Mean distance: (not calculated yet)
Parents
Sign in to add mentor| Ingrid H. Sarelius | grad student | 2008 | Rochester | |
| (ICAM-1 distribution in the microcirculation: Functional consequences for inflammatory responses.) | ||||
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Publications
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| Ren X, Manzanares LD, Piccolo EB, et al. (2023) Macrophage-endothelial cell crosstalk orchestrates neutrophil recruitment in inflamed mucosa. The Journal of Clinical Investigation |
| Rehring JF, Bui TM, Galán-Enríquez CS, et al. (2021) Released Myeloperoxidase Attenuates Neutrophil Migration and Accumulation in Inflamed Tissue. Frontiers in Immunology. 12: 654259 |
| Bui TM, Wiesolek HL, Sumagin R. (2020) ICAM-1: A master regulator of cellular responses in inflammation, injury resolution, and tumorigenesis. Journal of Leukocyte Biology |
| Wiesolek HL, Bui TM, Lee JJ, et al. (2020) ICAM-1 functions as an efferocytosis receptor in inflammatory macrophages. The American Journal of Pathology |
| Matthews JD, Owens JA, Naudin CR, et al. (2019) Neutrophil-derived reactive oxygen orchestrates epithelial cell signaling events during intestinal repair. The American Journal of Pathology |
| Bui TM, Sumagin R. (2019) Progressing from Recurring Tissue Injury to Genomic Instability: A New Mechanism of Neutrophil Pathogenesis. Dna and Cell Biology |
| Sullivan DP, Bui T, Muller WA, et al. (2018) In vivo imaging reveals unique neutrophil transendothelial migration patterns in inflamed intestines. Mucosal Immunology |
| Finkielsztein A, Mascarenhas L, Butin-Israeli V, et al. (2018) Isolation and Characterization of Neutrophil-derived Microparticles for Functional Studies. Journal of Visualized Experiments : Jove |
| Bui TM, Mascarenhas LA, Sumagin R. (2018) Extracellular vesicles regulate immune responses and cellular function in intestinal inflammation and repair. Tissue Barriers. e1431038 |
| Mei Y, Feng G, Rahimi N, et al. (2017) Loss of mDia1 causes neutropenia via attenuated CD11b endocytosis and increased neutrophil adhesion to the endothelium. Blood Advances. 1: 1650-1656 |