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Judd M. Aiken

Affiliations: 
University of Wisconsin, Madison, Madison, WI 
Area:
Cell Biology, Molecular Biology, General Biophysics
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Parents

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Children

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Volga A. Iniguez Rojas grad student 2000 UW Madison
Marisol E. Lopez grad student 2000 UW Madison
Jonathan Wanagat grad student 2000 UW Madison
Entela A. Bua grad student 2004 UW Madison
Allen J. Herbst grad student 2008 UW Madison
Laura R. Moody grad student 2010 UW Madison

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Publications

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Vandiver AR, Hoang AN, Herbst A, et al. (2023) Nanopore sequencing identifies a higher frequency and expanded spectrum of mitochondrial DNA deletion mutations in human aging. Aging Cell. e13842
Herbst A, Choi S, Hoang AN, et al. (2022) Remdesivir does not affect mitochondrial DNA copy number or deletion mutation frequency in aged male rats: A short report. Plos One. 17: e0271850
Herbst A, Aiken JM, Kim C, et al. (2022) Age- and time-dependent mitochondrial genotoxic and myopathic effects of beta-guanidinopropionic acid, a creatine analog, on rodent skeletal muscles. Geroscience
Herbst A, Wohlgemuth S, Yang J, et al. (2022) Susceptibility of Beavers to Chronic Wasting Disease. Biology. 11
O'Hara E, Herbst A, Kommadath A, et al. (2022) Neural transcriptomic signature of chronic wasting disease in white-tailed deer. Bmc Genomics. 23: 69
Herbst A, Hoang A, Kim C, et al. (2021) Metformin treatment in old rats and effects on mitochondrial integrity. Rejuvenation Research
Herbst A, Prior SJ, Lee CC, et al. (2021) Skeletal muscle mitochondrial DNA copy number and mitochondrial DNA deletion mutation frequency as predictors of physical performance in older men and women. Geroscience
Herbst A, Lee CC, Vandiver AR, et al. (2020) Mitochondrial DNA deletion mutations increase exponentially with age in human skeletal muscle. Aging Clinical and Experimental Research
Herbst A, Aiken JM, McKenzie D, et al. (2020) Comment on: "Mitochondrial Mechanisms of Neuromuscular Junction Degeneration with Aging. 2020, , 197". Cells. 9
Duque Velásquez C, Kim C, Haldiman T, et al. (2020) Chronic wasting disease (CWD) prion strains evolve via adaptive diversification of conformers in hosts expressing prion protein polymorphisms. The Journal of Biological Chemistry
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