Janice D. Wagner

Affiliations: 
Wake Forest University School of Medicine, Winston-Salem, NC, United States 
Area:
Pathology, Medical Biophysics, Genetics
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"Janice Wagner"
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Publications

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Kiguchi N, Ding H, Peters CM, et al. (2016) Altered expression of glial markers, chemokines, and opioid receptors in the spinal cord of type 2 diabetic monkeys. Biochimica Et Biophysica Acta
Morales-Corraliza J, Wong H, Mazzella MJ, et al. (2016) Brain-Wide Insulin Resistance, Tau Phosphorylation Changes, and Hippocampal Neprilysin and Amyloid-β Alterations in a Monkey Model of Type 1 Diabetes. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 36: 4248-58
Harwood HJ, Listrani P, Wagner JD. (2012) Nonhuman primates and other animal models in diabetes research. Journal of Diabetes Science and Technology. 6: 503-14
Dewi FN, Wood CE, Lees CJ, et al. (2012) Abstract 4298: In-utero, but not pubertal, soy exposure suppresses estrogen-regulated gene expression in non-human primate breast Cancer Research. 72: 4298-4298
Howard TD, Ho SM, Zhang L, et al. (2011) Epigenetic changes with dietary soy in cynomolgus monkeys. Plos One. 6: e26791
Kavanagh K, Flynn DM, Nelson C, et al. (2011) Characterization and validation of a streptozotocin-induced diabetes model in the vervet monkey. Journal of Pharmacological and Toxicological Methods. 63: 296-303
Gray SB, Langefeld CD, Ziegler JT, et al. (2011) Single-nucleotide polymorphisms in the TNF gene are associated with obesity-related phenotypes in vervet monkeys. Obesity (Silver Spring, Md.). 19: 1427-32
Kavanagh K, Flynn DM, Jenkins KA, et al. (2011) Restoring HSP70 deficiencies improves glucose tolerance in diabetic monkeys. American Journal of Physiology. Endocrinology and Metabolism. 300: E894-901
Rowe PA, Kavanagh K, Zhang L, et al. (2011) Short-term hyperglycemia increases arterial superoxide production and iron dysregulation in atherosclerotic monkeys. Metabolism: Clinical and Experimental. 60: 1070-80
Kavanagh K, Sajadian S, Jenkins KA, et al. (2010) Neonatal and fetal exposure to trans-fatty acids retards early growth and adiposity while adversely affecting glucose in mice. Nutrition Research (New York, N.Y.). 30: 418-26
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