Monica Venere, Ph.D.
Affiliations: | 2005 | University of Pennsylvania, Philadelphia, PA, United States |
Area:
Cell Biology, Molecular BiologyGoogle:
"Monica Venere"Mean distance: (not calculated yet)
Parents
Sign in to add mentor| Thanos D. Halazonetis | grad student | 2005 | Penn | |
| (Responding to DNA damage: Recruitment and activation of ATM and ATR.) | ||||
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Publications
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| Summers MK, Venere M. (2023) Walking the tightrope between DNA damage repair and genomic integrity. Neuro-Oncology |
| Venere M, Lathia JD. (2021) All Damage Is Not Created Equal: Unraveling the Complexity of Sex Chromosomes and Hormones in the DNA Damage Response. Endocrinology. 162 |
| Zalenski AA, Majumder S, De K, et al. (2020) An interphase pool of KIF11 localizes at the basal bodies of primary cilia and a reduction in KIF11 expression alters cilia dynamics. Scientific Reports. 10: 13946 |
| Pal D, Torres AE, Stromberg BR, et al. (2020) Chk1-mediated phosphorylation of Cdh1 promotes the SCF-dependent degradation of Cdh1 during S-phase and efficient cell-cycle progression. Cell Death & Disease. 11: 298 |
| De K, Grubb TM, Zalenski AA, et al. (2019) Hyperphosphorylation of CDH1 in glioblastoma cancer stem cells attenuates APC/CCDH1 activity and pharmacological inhibition of APC/CCDH1/CDC20 compromises viability. Molecular Cancer Research : McR |
| Jarrar A, Lotti F, DeVecchio J, et al. (2018) PARP inhibition sensitizes colorectal cancer-initiating cells to chemotherapy. Stem Cells (Dayton, Ohio) |
| Sharma A, Alswillah T, Singh K, et al. (2018) USP14 regulates DNA damage repair by targeting RNF168-dependent ubiquitination. Autophagy |
| Venere M. (2017) Self-tanning cells, the new SPF. Science Translational Medicine. 9 |
| Dermawan JK, Hitomi M, Silver DJ, et al. (2016) Pharmacological targeting of the histone chaperone complex FACT preferentially eliminates glioblastoma stem cells and prolongs survival in preclinical models. Cancer Research |
| Venere M, Hamerlik P, Wu Q, et al. (2014) Therapeutic targeting of constitutive PARP activation compromises stem cell phenotype and survival of glioblastoma-initiating cells. Cell Death and Differentiation. 21: 258-69 |