Neil Copes
Affiliations: | 2009-2015 | Cell Biology, Microbiology, and Molecular Biology | University of South Florida, Tampa, FL, United States |
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Parents
Sign in to add mentor| Patrick C. Bradshaw | grad student | 2009-2015 | University of South Florida |
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Publications
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| Ari C, Canfield CE, Copes N, et al. (2017) The Deanna protocol supplement complex supports mitochondrial energy metabolism and prolongs lifespan in preclinical models of amyotrophic lateral sclerosis (ALS) Metabolomics. 13 |
| Copes N, Edwards C, Chaput D, et al. (2015) Metabolome and proteome changes with aging in Caenorhabditis elegans. Experimental Gerontology. 72: 67-84 |
| Edwards C, Copes N, Bradshaw PC. (2015) D-ß-hydroxybutyrate: an anti-aging ketone body. Oncotarget. 6: 3477-8 |
| Edwards C, Canfield J, Copes N, et al. (2015) Mechanisms of amino acid-mediated lifespan extension in Caenorhabditis elegans. Bmc Genetics. 16: 8 |
| Edwards C, Canfield J, Copes N, et al. (2014) D-beta-hydroxybutyrate extends lifespan in C. elegans. Aging. 6: 621-44 |
| Edwards CB, Copes N, Brito AG, et al. (2013) Malate and fumarate extend lifespan in Caenorhabditis elegans. Plos One. 8: e58345 |
| Edwards CB, Copes N, Brito AG, et al. (2013) Malate treatment increased C. elegans NAD and NADPH levels more than succinate. Plos One |
| Dragicevic N, Delic V, Cao C, et al. (2012) Caffeine increases mitochondrial function and blocks melatonin signaling to mitochondria in Alzheimer's mice and cells. Neuropharmacology. 63: 1368-79 |
| Dragicevic N, Smith A, Lin X, et al. (2011) Green tea epigallocatechin-3-gallate (EGCG) and other flavonoids reduce Alzheimer's amyloid-induced mitochondrial dysfunction. Journal of Alzheimer's Disease : Jad. 26: 507-21 |
| Dragicevic N, Copes N, O'Neal-Moffitt G, et al. (2011) Melatonin treatment restores mitochondrial function in Alzheimer's mice: a mitochondrial protective role of melatonin membrane receptor signaling. Journal of Pineal Research. 51: 75-86 |