Collaborators
Sign in to add collaborator| Ian D. Forsythe | collaborator | Kings College London (Neurotree) |
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Publications
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| Spiers JG, Steinert JR. (2021) Nitrergic modulation of ion channel function in regulating neuronal excitability. Channels (Austin, Tex.). 15: 666-679 |
| Scheiblich H, Steinert JR. (2021) Nitrergic modulation of neuronal excitability in the mouse hippocampus is mediated via regulation of Kv2 and voltage-gated sodium channels. Hippocampus |
| Bourgognon JM, Spiers JG, Robinson SW, et al. (2021) Inhibition of neuroinflammatory nitric oxide signaling suppresses glycation and prevents neuronal dysfunction in mouse prion disease. Proceedings of the National Academy of Sciences of the United States of America. 118 |
| Mukaetova-Ladinska EB, Steinert J, Maltby J, et al. (2020) Further evidence for a role for the locus coeruleus in the aetiopathogenesis of dementia. The British Journal of Psychiatry : the Journal of Mental Science. 217: 652-653 |
| Amelio I, Panatta E, Niklison-Chirou MV, et al. (2020) The C terminus of p73 is essential for hippocampal development. Proceedings of the National Academy of Sciences of the United States of America |
| Choudhury N, Linley D, Richardson A, et al. (2020) Kv3.1 and Kv3.3 subunits differentially contribute to Kv3 channels and action potential repolarization in principal neurons of the auditory brainstem. The Journal of Physiology |
| Spiers JG, Breda C, Robinson S, et al. (2019) Nrf2/Keap1 Mediated Redox Signaling Supports Synaptic Function and Longevity and Impacts on Circadian Activity. Frontiers in Molecular Neuroscience. 12: 86 |
| Spiers JG, Chen HC, Bourgognon JM, et al. (2019) Dysregulation of stress systems and nitric oxide signaling underlies neuronal dysfunction in Alzheimer's disease. Free Radical Biology & Medicine |
| Bourgognon JM, Steinert JR. (2019) The metabolome identity: basis for discovery of biomarkers in neurodegeneration. Neural Regeneration Research. 14: 387-390 |
| Bourgognon JM, Spiers JG, Scheiblich H, et al. (2018) Alterations in neuronal metabolism contribute to the pathogenesis of prion disease. Cell Death and Differentiation |