Thomas Lahaye
Affiliations: | Department of Biology | Martin Luther University Halle-Wittenberg, Germany |
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"Thomas Lahaye"Children
Sign in to add trainee| Sebastian Schornack | grad student | 2001-2006 | Martin Luther University Halle-Wittenberg |
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Publications
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| Gallas N, Li X, von Roepenack-Lahaye E, et al. (2023) An ancient cis-element targeted by Ralstonia solanacearum TALE-like effectors facilitates the development of a promoter trap that could confer broad-spectrum wilt resistance. Plant Biotechnology Journal |
| Shantharaj D, Minsavage GV, Orbović V, et al. (2023) A promoter trap in transgenic citrus mediates recognition of a broad spectrum of Xanthomonas citri pv. citri TALEs, including in planta-evolved derivatives. Plant Biotechnology Journal |
| You Y, Koczyk G, Nuc M, et al. (2022) The eINTACT system dissects bacterial exploitation of plant osmosignalling to enhance virulence. Nature Plants |
| Schenstnyi K, Strauß A, Dressel A, et al. (2022) The tomato resistance gene Bs4 suppresses leaf watersoaking phenotypes induced by AvrHah1, a transcription activator-like effector from tomato-pathogenic xanthomonads. The New Phytologist |
| Nowack MK, Holmes DR, Lahaye T. (2021) TALE-induced cell death executors: an origin outside immunity? Trends in Plant Science |
| Krönauer C, Lahaye T. (2021) The flavin monooxygenase Bs3 triggers cell death in plants, impairs growth in yeast and produces H2O2 in vitro. Plos One. 16: e0256217 |
| Wu D, von Roepenack-Lahaye E, Buntru M, et al. (2019) A Plant Pathogen Type III Effector Protein Subverts Translational Regulation to Boost Host Polyamine Levels. Cell Host & Microbe |
| Krönauer C, Kilian J, Strauß T, et al. (2019) Cell death triggered by the YUCCA-like Bs3 protein coincides with accumulation of SA and Pip but not of IAA. Plant Physiology |
| Krönauer C, Kilian J, Strauß T, et al. (2019) Cell death triggered by the YUCCA-like Bs3 protein coincides with accumulation of SA and Pip but not of IAA. Plant Physiology |
| de Lange O, Schandry N, Wunderlich M, et al. (2017) Exploiting the sequence diversity of TALE-like repeats to vary the strength of dTALE-promoter interactions. Synthetic Biology (Oxford, England). 2: ysx004 |