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Massimo Tabaton

Affiliations: 
University of Genoa, Genova, Liguria, Italy 
Area:
Alzheimer disease
Website:
http://frontiersin.org/profiles/massimotabaton/
Google:
"Massimo Tabaton"
Bio:

http://www.alzforum.org/com/res/detail.asp?id={92EDD761-D1C4-4B0E-A53E-23B271A14129}

Cross-listing: Neurotree - Neuropathology Tree

BETA: Related publications

Publications

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Guglielmotto M, Manassero G, Vasciaveo V, et al. (2020) Estrogens Inhibit Amyloid-β-Mediated Paired Helical Filament-Like Conformation of Tau Through Antioxidant Activity and miRNA 218 Regulation in hTau Mice. Journal of Alzheimer's Disease : Jad
Bongianni M, Ladogana A, Capaldi S, et al. (2019) α-Synuclein RT-QuIC assay in cerebrospinal fluid of patients with dementia with Lewy bodies. Annals of Clinical and Translational Neurology
Guglielmotto M, Repetto IE, Monteleone D, et al. (2019) Stroke and Amyloid-β Downregulate TREM-2 and Uch-L1 Expression that Synergistically Promote the Inflammatory Response. Journal of Alzheimer's Disease : Jad
Monacelli F, Martella L, Parodi MN, et al. (2019) Frontal Variant of Alzheimer's Disease: A Report of a Novel PSEN1 Mutation. Journal of Alzheimer's Disease : Jad
Testa G, Staurenghi E, Giannelli S, et al. (2018) A silver lining for 24-hydroxycholesterol in Alzheimer's disease: The involvement of the neuroprotective enzyme sirtuin 1. Redox Biology. 17: 423-431
Perry G, Avila J, Moreira PI, et al. (2018) Preface. Journal of Alzheimer's Disease : Jad
Tamagno E, Guglielmotto M, Monteleone D, et al. (2017) The Unexpected Role of Aβ1-42 Monomers in the Pathogenesis of Alzheimer's Disease. Journal of Alzheimer's Disease : Jad
Guglielmotto M, Monteleone D, Vasciaveo V, et al. (2017) The Decrease of Uch-L1 Activity Is a Common Mechanism Responsible for Aβ 42 Accumulation in Alzheimer's and Vascular Disease. Frontiers in Aging Neuroscience. 9: 320
Manassero G, Guglielmotto M, Monteleone D, et al. (2017) Dual Mechanism of Toxicity for Extracellular Injection of Tau Oligomers versus Monomers in Human Tau Mice. Journal of Alzheimer's Disease : Jad
Manassero G, Guglielmotto M, Zamfir R, et al. (2016) Beta-amyloid 1-42 monomers, but not oligomers, produce PHF-like conformation of Tau protein. Aging Cell
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