Dora Kovacs, PhD

Neurology Massachusetts General Hospital and Harvard University, Boston, MA, United States 
molecular events underlying neurodegeneration in aging, and particularly in Alzheimer’s disease
"Dora Kovacs"
Cross-listing: Neurotree - Neuropathology Tree

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Bhattacharyya R, Fenn RH, Barren C, et al. (2016) Palmitoylated APP Forms Dimers, Cleaved by BACE1. Plos One. 11: e0166400
Kuzuya A, Zoltowska KM, Post KL, et al. (2016) Identification of the novel activity-driven interaction between synaptotagmin 1 and presenilin 1 links calcium, synapse, and amyloid beta. Bmc Biology. 14: 25
Gautam V, D'Avanzo C, Berezovska O, et al. (2015) Synaptotagmins interact with APP and promote Aβ generation. Molecular Neurodegeneration. 10: 31
D'Avanzo C, Sliwinski C, Wagner SL, et al. (2015) γ-Secretase modulators reduce endogenous amyloid β42 levels in human neural progenitor cells without altering neuronal differentiation. Faseb Journal : Official Publication of the Federation of American Societies For Experimental Biology
Choi SH, Kim YH, Hebisch M, et al. (2014) A three-dimensional human neural cell culture model of Alzheimer's disease. Nature. 515: 274-8
Mótyán G, Kádár Z, Kovács D, et al. (2014) Regio- and stereoselective access to novel ring-condensed steroidal isoxazolines. Steroids. 87: 76-85
Frank É, Kovács D, Schneider G, et al. (2014) Synthesis of novel steroidal 16-spiroisoxazolines by 1,3-dipolar cycloaddition, and an evaluation of their antiproliferative activities in vitro. Molecular Diversity. 18: 521-34
Kovács D, Mótyán G, Wölfling J, et al. (2014) A facile access to novel steroidal 17-2'-(1',3',4')-oxadiazoles, and an evaluation of their cytotoxic activities in vitro. Bioorganic & Medicinal Chemistry Letters. 24: 1265-8
Gautam V, D'Avanzo C, Hebisch M, et al. (2014) BACE1 activity regulates cell surface contactin-2 levels. Molecular Neurodegeneration. 9: 4
Kim DY, Wertz MH, Gautam V, et al. (2014) The E280A presenilin mutation reduces voltage-gated sodium channel levels in neuronal cells. Neuro-Degenerative Diseases. 13: 64-8
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